实验性自身免疫性重症肌无力中的淋巴细胞激活 - Suppr

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超能文献

Lymphocyte activation in experimental autoimmune myasthenia gravis.

作者信息

De Baets M H, Einarson B, Lindstrom J M, Weigle W O

出版信息

J Immunol. 1982 May;128(5):2228-35.

PMID:6977589

Abstract

摘要

相似文献

Lymphocyte activation in experimental autoimmune myasthenia gravis.实验性自身免疫性重症肌无力中的淋巴细胞激活

J Immunol. 1982 May;128(5):2228-35.

Genetic control of experimental autoimmune myasthenia gravis in mice. I. Lymphocyte proliferative response to acetylcholine receptors is under H-2-linked Ir gene control.小鼠实验性自身免疫性重症肌无力的遗传控制。I. 对乙酰胆碱受体的淋巴细胞增殖反应受H-2连锁免疫反应基因控制。

J Immunol. 1979 Dec;123(6):2540-3.

Oral administration of acetylcholine receptor: effects on experimental myasthenia gravis.口服乙酰胆碱受体：对实验性重症肌无力的影响。

Ann Neurol. 1994 Nov;36(5):704-13. doi: 10.1002/ana.410360504.

Lymphocyte responsiveness to acetylcholine receptor in rats with experimental autoimmune myasthenia gravis.实验性自身免疫性重症肌无力大鼠中淋巴细胞对乙酰胆碱受体的反应性

Ann Neurol. 1980 Jul;8(1):32-7. doi: 10.1002/ana.410080105.

T cell reactivity to acetylcholine receptor in rats orally tolerized against experimental autoimmune myasthenia gravis.对经口服耐受实验性自身免疫性重症肌无力的大鼠中乙酰胆碱受体的T细胞反应性。

Cell Immunol. 1993 Dec;152(2):394-404. doi: 10.1006/cimm.1993.1300.

Inhibition of alpha-bungarotoxin binding to acetylcholine receptors by antisera from animals with experimental autoimmune myasthenia gravis.实验性自身免疫性重症肌无力动物抗血清对α-银环蛇毒素与乙酰胆碱受体结合的抑制作用。

J Supramol Struct. 1980;14(3):267-79. doi: 10.1002/jss.400140302.

Experimental autoimmune myasthenia gravis may occur in the context of a polarized Th1- or Th2-type immune response in rats.实验性自身免疫性重症肌无力可能发生在大鼠极化的Th1型或Th2型免疫反应的背景下。

J Immunol. 1999 Jun 15;162(12):7189-97.

B-cell activation in vitro by helper T cells specific to region alpha 146-162 of Torpedo californica nicotinic acetylcholine receptor.由加州电鳐烟碱型乙酰胆碱受体α146 - 162区域特异性辅助性T细胞介导的体外B细胞活化。

J Immunol. 1996 Oct 1;157(7):3192-9.

Postsynapic membrane of Torpedo electroplax: acetylcholine receptor localization and its use as a tool in the study of cellular immuninty in myasthenia gravis.电鳐电器官的突触后膜：乙酰胆碱受体定位及其在重症肌无力细胞免疫研究中的应用

Adv Cytopharmacol. 1979;3:255-67.

The limitation of IL-10-exposed dendritic cells in the treatment of experimental autoimmune myasthenia gravis and myasthenia gravis.白细胞介素-10处理的树突状细胞在实验性自身免疫性重症肌无力和重症肌无力治疗中的局限性。

Cell Immunol. 2006 Jun;241(2):95-101. doi: 10.1016/j.cellimm.2006.08.005. Epub 2006 Sep 26.

引用本文的文献

Standardization of the experimental autoimmune myasthenia gravis (EAMG) model by immunization of rats with Torpedo californica acetylcholine receptors--Recommendations for methods and experimental designs.通过用加州电鳐乙酰胆碱受体免疫大鼠对实验性自身免疫性重症肌无力（EAMG）模型进行标准化——方法和实验设计建议

Exp Neurol. 2015 Aug;270:18-28. doi: 10.1016/j.expneurol.2015.03.010. Epub 2015 Mar 18.

A comparison of epitope repertoires associated with myasthenia gravis in humans and nonhuman hosts.人类和非人类宿主中与重症肌无力相关的表位库比较。

Autoimmune Dis. 2012;2012:403915. doi: 10.1155/2012/403915. Epub 2012 Dec 2.

Myasthenia gravis as a prototype autoimmune receptor disease.重症肌无力作为自身免疫性受体疾病的典型代表。

Immunol Res. 1997;16(4):341-54. doi: 10.1007/BF02786398.

Myasthenia gravis induces the activation and maturation of lymphocytes in thymoma.重症肌无力可诱导胸腺瘤中淋巴细胞的活化和成熟。

J Clin Immunol. 1996 Jul;16(4):190-7. doi: 10.1007/BF01541224.

Inhibition of T-cell reactivity to myasthenogenic epitopes of the human acetylcholine receptor by synthetic analogs.合成类似物对人乙酰胆碱受体致肌无力表位的T细胞反应性的抑制作用。

Proc Natl Acad Sci U S A. 1993 Aug 1;90(15):7000-4. doi: 10.1073/pnas.90.15.7000.

Myasthenia gravis: an autoimmune response against the acetylcholine receptor.重症肌无力：针对乙酰胆碱受体的自身免疫反应。

Immunol Res. 1993;12(1):78-100. doi: 10.1007/BF02918370.

Immunopathology of acetylcholine receptors in myasthenia gravis.重症肌无力中乙酰胆碱受体的免疫病理学

Springer Semin Immunopathol. 1982;5(4):389-412. doi: 10.1007/BF01857427.

Progress on studies of myasthenia gravis during 1982.1982年重症肌无力的研究进展

Surv Immunol Res. 1983;2(3):293-5. doi: 10.1007/BF02918435.

In vivo therapy with monoclonal anti-I-A antibody suppresses immune responses to acetylcholine receptor.用单克隆抗I-A抗体进行体内治疗可抑制对乙酰胆碱受体的免疫反应。

Proc Natl Acad Sci U S A. 1983 May;80(9):2713-7. doi: 10.1073/pnas.80.9.2713.

Induction of acetylcholine receptor-specific suppression. An in vitro model of antigen-specific immunosuppression in myasthenia gravis.诱导乙酰胆碱受体特异性抑制。重症肌无力抗原特异性免疫抑制的体外模型。

J Exp Med. 1984 Jan 1;159(1):292-304. doi: 10.1084/jem.159.1.292.

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