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毒毛花苷K或乙酰胆碱诱导挛缩过程中组织氧化还原状态电位与[K⁺]₀活性的dak/dt变化之间的关系。

Relationship between the tissue redox state potential and dak/dt changes of [K+]0 activity during k-strophantoside or acetylcholine induced contractures.

作者信息

Wittmann I, Puppi A, Dely M

出版信息

Acta Physiol Acad Sci Hung. 1982;60(4):233-6.

PMID:6985315
Abstract

It was established that oxidosis evoked by methylene blue during k-strophantoside and acetylcholine contractures in skeletal muscle was accompanied by an increase in the changes of [K+]0 activity, while redosis evoked by ascorbate under the same conditions was accompanied by a decrease in the changes of [K+]0 activity. These changes in [K+]0 activity parallelled the alterations of contractures caused by oxidosis or redosis suggesting that changes in the [K+]0 transient play an important role in these phenomena.

摘要

已经确定,在骨骼肌中,毒毛花苷K和乙酰胆碱挛缩期间亚甲蓝诱发的氧化中毒伴随着[K⁺]₀活性变化的增加,而在相同条件下抗坏血酸诱发的还原中毒则伴随着[K⁺]₀活性变化的减少。[K⁺]₀活性的这些变化与氧化中毒或还原中毒引起的挛缩改变平行,这表明[K⁺]₀瞬变的变化在这些现象中起重要作用。

相似文献

1
Relationship between the tissue redox state potential and dak/dt changes of [K+]0 activity during k-strophantoside or acetylcholine induced contractures.毒毛花苷K或乙酰胆碱诱导挛缩过程中组织氧化还原状态电位与[K⁺]₀活性的dak/dt变化之间的关系。
Acta Physiol Acad Sci Hung. 1982;60(4):233-6.
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