Blomlöf L, Otteskog P, Söder P O
Scand J Dent Res. 1980 Feb;88(1):10-4. doi: 10.1111/j.1600-0722.1980.tb00713.x.
The interaction of LPS from Bacteroides fragilis, Escherichia coli, Salmonella typhimurium and lipid A from S. typhimurium with adhesion of human periodontal cells on plastic surfaces has been studied. The results showed that LPS and lipid A reduced adhesion already after 5 min at concentrations from 5 to 200 microgram/ml without influencing cell viability. It could be demonstrated that LPS and lipid A interact with both the cell and the substratum surface. Lipid A from S. typhimurium was more potent than any of the LPS used.
研究了脆弱拟杆菌、大肠杆菌、鼠伤寒沙门氏菌的脂多糖（LPS）以及鼠伤寒沙门氏菌的脂质A与人牙周细胞在塑料表面黏附的相互作用。结果表明，在浓度为5至200微克/毫升时，LPS和脂质A在5分钟后就降低了细胞黏附，且不影响细胞活力。可以证明，LPS和脂质A与细胞及基质表面均有相互作用。鼠伤寒沙门氏菌的脂质A比所使用的任何一种LPS的作用都更强。
