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The effect of superoxide dismutase, catalase and L-cysteine on spontaneous and on mitomycin C induced chromosomal breakage in Fanconi's anemia and normal fibroblasts as measured by the micronucleus method.

作者信息

Raj A S, Heddle J A

出版信息

Mutat Res. 1980 May;78(1):59-66. doi: 10.1016/0165-1218(80)90026-9.

DOI:10.1016/0165-1218(80)90026-9
PMID:6991929
Abstract
摘要

相似文献

1
The effect of superoxide dismutase, catalase and L-cysteine on spontaneous and on mitomycin C induced chromosomal breakage in Fanconi's anemia and normal fibroblasts as measured by the micronucleus method.通过微核法测定超氧化物歧化酶、过氧化氢酶和L-半胱氨酸对范科尼贫血和成纤维细胞自发的以及丝裂霉素C诱导的染色体断裂的影响。
Mutat Res. 1980 May;78(1):59-66. doi: 10.1016/0165-1218(80)90026-9.
2
Effect of superoxide dismutase and catalase on spontaneously occurring chromosome breaks in patients with Fanconi's anemia.
Hereditas. 1977;86(2):147-50. doi: 10.1111/j.1601-5223.1977.tb01223.x.
3
Suppression of spontaneous and mitomycin C-induced chromosome aberrations in Fanconi's anemia by cell fusion with normal human fibroblasts.通过与正常人成纤维细胞进行细胞融合抑制范科尼贫血中自发的和丝裂霉素C诱导的染色体畸变。
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4
Sensitivity to five mutagens in Fanconi's anemia as measured by the micronucleus method.
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5
Fanconi's anemia--chromosome breakage studies in homozygotes and heterozygotes.范科尼贫血——纯合子和杂合子的染色体断裂研究
Cancer Genet Cytogenet. 1988 Jul 15;33(2):175-83. doi: 10.1016/0165-4608(88)90027-1.
6
Clinical and cytogenetic diversity in Fanconi's anaemia.范可尼贫血的临床和细胞遗传学多样性
J Med Genet. 1984 Jun;21(3):197-203. doi: 10.1136/jmg.21.3.197.
7
Complementation studies between Fanconi's anemia cells with different DNA repair characteristics.具有不同DNA修复特征的范可尼贫血细胞之间的互补研究。
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9
Antagonistic effect of cocultivation on mitomycin C-induced aberration rate in cells of a patient with Fanconi's anemia and in Chinese hamster ovary cells.共培养对范可尼贫血患者细胞及中国仓鼠卵巢细胞中丝裂霉素C诱导的畸变率的拮抗作用。
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Analysis of heterogeneity in Fanconi's anemia patients of different ethnic origin.不同种族范科尼贫血患者的异质性分析。
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Cytoprotective effect of honey against chromosomal breakage in fanconi anemia patients in vitro.蜂蜜对范科尼贫血患者体外染色体断裂的细胞保护作用。
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3
Amifostine protection against mitomycin-induced chromosomal breakage in fanconi anaemia lymphocytes.
氨磷汀对丝裂霉素诱导的范科尼贫血淋巴细胞染色体断裂的保护作用。
Molecules. 2008 Aug 21;13(8):1759-72. doi: 10.3390/molecules13081759.
4
Oxidative stress-related mechanisms are associated with xenobiotics exerting excess toxicity to Fanconi anemia cells.氧化应激相关机制与外源性物质对范可尼贫血细胞产生过度毒性有关。
Environ Health Perspect. 2003 Nov;111(14):1699-703. doi: 10.1289/ehp.6229.
5
Enhancement of sister-chromatid exchanges by tumour promoters.肿瘤启动子对姐妹染色单体交换的增强作用。
Br J Cancer. 1982 May;45(5):769-77. doi: 10.1038/bjc.1982.119.
6
Proliferative kinetics and mitomycin C-induced chromosome damage in Fanconi's anemia lymphocytes.范科尼贫血淋巴细胞的增殖动力学及丝裂霉素C诱导的染色体损伤
Hum Genet. 1983;63(1):19-23. doi: 10.1007/BF00285391.
7
Effect of oxidants and antioxidants on chromosomal breakage in Fanconi anemia lymphocytes.氧化剂和抗氧化剂对范可尼贫血淋巴细胞染色体断裂的影响。
Hum Genet. 1985;69(1):62-5. doi: 10.1007/BF00295530.
8
Bovine superoxide dismutase in Fanconi anaemia. Therapeutic trial in two patients.范可尼贫血中的牛超氧化物歧化酶。两名患者的治疗试验。
Hum Genet. 1985;70(2):181-2. doi: 10.1007/BF00273079.
9
Antioxidant status of Fanconi anemia fibroblasts.范可尼贫血成纤维细胞的抗氧化状态。
Hum Genet. 1987 Sep;77(1):28-31. doi: 10.1007/BF00284708.
10
Fanconi anemia mutation causes cellular susceptibility to ambient oxygen.范可尼贫血突变导致细胞对环境氧的易感性。
Am J Hum Genet. 1988 Oct;43(4):429-35.