Przybojewski J Z, Barnard P M
S Afr Med J. 1981 Feb 7;59(6):190-8.
A fairly young man suddenly developed a large ventricular septal defect (VSD) after an extensive anterolateral and anteroseptal myocardial infarction. Because of congestive cardiac failure refractory to medical treatment he underwent simultaneous closure of the VSD and left ventricular aneurysmectomy 6 weeks after the myocardial infarction. Some 14 months after operation the patient is completely asymptomatic on the minimum of medical therapy and is able to live normally. The literature on these two combined lesions complicating acute myocardial infarction and their surgical correction was caused by coronary spasm of the Prinzmetal variety with underlying normal coronary arteries.
一名相当年轻的男子在广泛的前外侧和前间隔心肌梗死后突然出现了一个大的室间隔缺损（VSD）。由于药物治疗难以控制的充血性心力衰竭，他在心肌梗死后6周接受了室间隔缺损封堵术和左心室室壁瘤切除术。术后约14个月，患者在接受最少药物治疗的情况下完全无症状，能够正常生活。关于这两种并发于急性心肌梗死的联合病变及其手术矫正的文献是由原发性冠状动脉痉挛伴潜在正常冠状动脉引起的。
