Haller H, Jaross W, Hanefeld M
Z Gesamte Inn Med. 1981 Aug 15;36(16):552-7.
Alcohol has a direct and/or indirect influence on the fat metabolism, evoking a HLP and this again may lead to secondary defects. Liver defects, pancreatitis, haemolytic conditions may be the sequel of the alcohol as well as of the HLP. When a HLP, particularly of type IV. is present, these statements demand a careful alcohol anamnesis. A control after absolute alcohol abstinence is necessary. If within a few days the HLP shows tendencies to involution, the alcohol can be proved as pathogenetic factor. The exclusion of the noxa is of decisive therapeutic importance and often spares a little effective and loaded with side-effects pharmacotherapy.
酒精对脂肪代谢有直接和/或间接影响，引发高脂血症（HLP），而这又可能导致继发性缺陷。肝脏缺陷、胰腺炎、溶血性疾病可能是酒精以及高脂血症的后果。当存在高脂血症，尤其是IV型高脂血症时，这些情况需要仔细询问饮酒史。绝对戒酒之后进行复查是必要的。如果在几天内高脂血症显示出消退的趋势，那么酒精可被证明是致病因素。排除致病因素具有决定性的治疗意义，并且常常可以避免使用效果欠佳且副作用大的药物治疗。
