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Inhibition of marker influx into complement-treated resealed erythrocyte ghosts by anti-C5.

作者信息

Simone C B, Henkart P

出版信息

J Immunol. 1982 Mar;128(3):1168-75.

PMID:7035559
Abstract
摘要

相似文献

1
Inhibition of marker influx into complement-treated resealed erythrocyte ghosts by anti-C5.
J Immunol. 1982 Mar;128(3):1168-75.
2
The functional size of the primary complement lesion in resealed erythrocyte membrane ghosts.
J Immunol. 1979 Jan;122(1):240-5.
3
Determination of the number of lytic sites in biconcave and spheroid erythrocyte ghosts after complement lysis.补体溶解后双凹形和球形红细胞血影中溶细胞位点数量的测定。
J Immunol. 1979 May;122(5):2032-6.
4
Complement-induced modifications in membrane fluidity: studies with resealed and glutaraldehyde-treated erythrocyte membrane ghosts.
J Immunol. 1978 Jun;120(6):2003-7.
5
Life-span and size of the trans-membrane channel formed by large doses of complement.大剂量补体形成的跨膜通道的寿命和大小。
J Immunol. 1980 May;124(5):2281-7.
6
Restriction of complement-mediated membrane damage by the eighth component of complement: a dual role for C8 in the complement attack sequence.补体第八成分对补体介导的膜损伤的限制作用:C8在补体攻击序列中的双重作用
J Immunol. 1979 Sep;123(3):1245-52.
7
The attack phase of human complement: differentiation between membrane binding and complex formation by the detection of neoantigen expression in situ. A morphometric immunoferritin study.人类补体的攻击阶段:通过原位检测新抗原表达区分膜结合与复合物形成。一项形态计量免疫铁蛋白研究。
J Immunol. 1981 Sep;127(3):1089-93.
8
Interaction between the labile binding sites of the fourth (C4) and fifth (C5) human complement proteins and erythrocyte cell membranes.人补体第四成分(C4)和第五成分(C5)的不稳定结合位点与红细胞细胞膜之间的相互作用。
J Immunol. 1980 Aug;125(2):634-9.
9
Analysis of solute diffusion across the C5b-9 membrane lesion of complement: evidence that individual C5b-9 complexes do not function as discrete, uniform pores.补体C5b-9膜损伤处溶质扩散分析:单个C5b-9复合物并非作为离散、均匀的孔发挥作用的证据。
J Immunol. 1980 Dec;125(6):2617-25.
10
On the mechanism of membrane damage by complement: the effect of length and unsaturation of the acyl chains in liposomal bilayers and the effect of cholesterol concentration in sheep erythrocyte and liposomal membranes.关于补体对膜损伤的机制:脂质体双层中酰基链长度和不饱和度的影响以及绵羊红细胞膜和脂质体膜中胆固醇浓度的影响。
J Immunol. 1978 Jun;120(6):1996-2002.

引用本文的文献

1
The transient pore formed by homologous terminal complement complexes functions as a bidirectional route for the transport of autocrine and paracrine signals across human cell membranes.由同源末端补体复合物形成的瞬时孔道作为自分泌和旁分泌信号跨人类细胞膜运输的双向通道。
Mol Med. 1996 Nov;2(6):755-65.
2
Effects of temperature, time, and toxin concentration on lesion formation by the Escherichia coli hemolysin.温度、时间和毒素浓度对大肠杆菌溶血素形成损伤的影响。
Infect Immun. 1994 Oct;62(10):4124-34. doi: 10.1128/iai.62.10.4124-4134.1994.
3
Is the membrane attack complex of complement an enzyme?
补体的膜攻击复合物是一种酶吗?
Mol Cell Biochem. 1984;61(1):5-15. doi: 10.1007/BF00239603.