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Defect in macrophage effector function confers Salmonella typhimurium susceptibility on C3H/HeJ mice.

作者信息

O'Brien A D, Metcalf E S, Rosenstreich D L

出版信息

Cell Immunol. 1982 Mar 1;67(2):325-33. doi: 10.1016/0008-8749(82)90224-6.

DOI:10.1016/0008-8749(82)90224-6
PMID:7044573
Abstract
摘要

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1
Defect in macrophage effector function confers Salmonella typhimurium susceptibility on C3H/HeJ mice.巨噬细胞效应功能缺陷使C3H/HeJ小鼠对鼠伤寒沙门氏菌易感。
Cell Immunol. 1982 Mar 1;67(2):325-33. doi: 10.1016/0008-8749(82)90224-6.
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Cellular immunity induced by avirulent Salmonella in LPS-defective C3H/HeJ mice.无毒力沙门氏菌在LPS缺陷型C3H/HeJ小鼠中诱导的细胞免疫。
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Evidence for separate genetic defects in C3H/HeJ and C3HeB/FeJ mice, that affect susceptibility to gram-negative infections.C3H/HeJ和C3HeB/FeJ小鼠中存在影响革兰氏阴性菌感染易感性的不同遗传缺陷的证据。
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Intestinal Lamina Propria CD4 T Cells Promote Bactericidal Activity of Macrophages via Galectin-9 and Tim-3 Interaction during Salmonella enterica Serovar Typhimurium Infection.肠固有层 CD4 T 细胞通过沙门氏菌感染期间半乳糖凝集素-9 和 Tim-3 的相互作用促进巨噬细胞的杀菌活性。
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The immunology of antituberculous immunity.抗结核免疫的免疫学
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IFNγ expression by an attenuated strain of Salmonella enterica serovar Typhimurium improves vaccine efficacy in susceptible TLR4-defective C3H/HeJ mice.减毒鼠伤寒沙门氏菌 Typhimurium 株 IFNγ 的表达增强了易感 TLR4 缺陷型 C3H/HeJ 小鼠疫苗的效力。
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