Torack R M
Comput Radiol. 1982 Jan-Feb;6(1):35-41. doi: 10.1016/0730-4862(82)90179-2.
An index case and 13 other cases of acute ischemic cerebral infarction have been examined following the occurrence of death 1-6 days postictus. Histologic studies and water content assay involved both the infarct and peri-infarct tissue. The primary site of fluid accumulation was the infarcted white matter; however, after 3 days, edema in the adjacent white matter may be significant. The reduced attenuation of CT scans is caused only by edema since necrosis of the white matter is not present. Reactive vascular changes that occur in the cortex appear to be the basis of early contrast enhancement. Three different mechanisms of edema formation have been proposed to be involved in the mass effect.
对1例索引病例和其他13例急性缺血性脑梗死病例在发病后1 - 6天死亡时进行了检查。组织学研究和含水量测定涉及梗死灶和梗死灶周围组织。液体蓄积的主要部位是梗死的白质；然而，3天后，相邻白质的水肿可能会很明显。CT扫描衰减降低仅是由水肿引起的，因为不存在白质坏死。皮质中发生的反应性血管变化似乎是早期强化的基础。已提出三种不同的水肿形成机制与占位效应有关。
