引发豚鼠腹腔巨噬细胞呼吸爆发的酶。 - Suppr

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The enzyme responsible for the respiratory burst in elicited guinea pig peritoneal macrophages.

作者信息

Berton G, Bellavite P, Dri P, de Togni P, Rossi F

出版信息

J Pathol. 1982 Apr;136(4):273-90. doi: 10.1002/path.1711360403.

DOI:10.1002/path.1711360403

PMID:7077433

Abstract

摘要

相似文献

The enzyme responsible for the respiratory burst in elicited guinea pig peritoneal macrophages.引发豚鼠腹腔巨噬细胞呼吸爆发的酶。

J Pathol. 1982 Apr;136(4):273-90. doi: 10.1002/path.1711360403.

Enzymatic basis of the respiratory burst of guinea pig resident peritoneal macrophages.

J Reticuloendothel Soc. 1981 Jan;29(1):47-60.

Molecular basis for the enhanced respiratory burst of activated macrophages.活化巨噬细胞呼吸爆发增强的分子基础。

Fed Proc. 1985 Nov;44(14):2927-32.

Activation of the respiratory burst of guinea pig neutrophils by dicyclohexylcarbodiimide.

FEBS Lett. 1983 May 8;155(2):205-8. doi: 10.1016/0014-5793(82)80603-0.

An assay for macrophage activating factor based on the adherence of oil-elicited guinea pig macrophages: characterization of a lymphokine-induced release of hydrogen peroxide from elicited macrophages.基于油诱导豚鼠巨噬细胞黏附的巨噬细胞激活因子测定：淋巴因子诱导的诱导巨噬细胞释放过氧化氢的特性

Lymphokine Res. 1986 Winter;5(1):35-47.

In vitro NADH-oxidase, NADPH-oxidase and myeloperoxidase activity of macrophages after Tinospora cordifolia (guduchi) treatment.钩藤（guduchi）处理后巨噬细胞的体外 NADH-氧化酶、NADPH-氧化酶和髓过氧化物酶活性。

Immunopharmacol Immunotoxicol. 2012 Jun;34(3):368-72. doi: 10.3109/08923973.2011.606324. Epub 2012 Feb 2.

Activation of mouse peritoneal macrophages by lipopolysaccharide alters the kinetic parameters of the superoxide-producing NADPH oxidase.脂多糖激活小鼠腹腔巨噬细胞会改变产生超氧化物的NADPH氧化酶的动力学参数。

J Biol Chem. 1983 Aug 25;258(16):9631-5.

Plasma membrane and phagosome localisation of the activated NADPH oxidase in elicited peritoneal macrophages of the guinea-pig.

J Pathol. 1982 Mar;136(3):241-52. doi: 10.1002/path.1711360307.

Production of active oxygen species by phagocytic leukocytes.吞噬性白细胞产生活性氧物质。

Immunol Ser. 1994;60:159-78.

Rat macrophage treatment with lipopolysaccharide leads to a reduction in respiratory burst product secretion and a decrease in NADPH oxidase affinity.用脂多糖处理大鼠巨噬细胞会导致呼吸爆发产物分泌减少以及NADPH氧化酶亲和力降低。

Cell Immunol. 1987 Aug;108(1):109-19. doi: 10.1016/0008-8749(87)90197-3.

引用本文的文献

Multianalyte Microphysiometry of Macrophage Responses to Phorbol Myristate Acetate, Lipopolysaccharide, and Lipoarabinomannan.巨噬细胞对佛波醇肉豆蔻酸酯乙酸酯、脂多糖和脂阿拉伯甘露聚糖反应的多分析物微生理测定

Electroanalysis. 2013 Jul 1;25(7):1706-1712. doi: 10.1002/elan.201300121.

The mechanism of action of lymphokines. VIII. Lymphokine-enhanced spontaneous hydrogen peroxide production by macrophages.淋巴因子的作用机制。VIII. 淋巴因子增强巨噬细胞自发产生过氧化氢的能力。

Immunology. 1985 Jan;54(1):35-45.

Activation of mouse macrophages causes no change in expression and function of phorbol diesters' receptors, but is accompanied by alterations in the activity and kinetic parameters of NADPH oxidase.小鼠巨噬细胞的激活不会导致佛波酯受体的表达和功能发生变化，但会伴随着NADPH氧化酶的活性和动力学参数的改变。

Immunology. 1985 Feb;54(2):371-9.

Cyclosporin A inhibits phorbol ester-induced activation of superoxide production in resident mouse peritoneal macrophages.环孢菌素A抑制佛波酯诱导的驻留小鼠腹膜巨噬细胞中超氧化物生成的激活。

Biochem J. 1989 Nov 15;264(1):21-6. doi: 10.1042/bj2640021.

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