McAfee P C, Yuan H A
Clin Orthop Relat Res. 1982 Jun(166):62-71.
Fourth generation computed tomography (CT) diagnostic studies were used to investigate or localize, neurologic changes and to clarify the anatomy of slippage in a consecutive series of 31 patients who had spondylolisthesis. Eighteen patients had operative procedures, i.e, either spinal fusions, decompressions, or both, after the CT examinations, and the average follow-up was 25 months. Ten postoperative CT studies were performed to document the correction of the original pathology. Fifteen patients had objective neurologic deficits referable to the spondylolisthesis, of whom CT demonstrated the specific sites of neural encroachment in 13. Preoperative myelography was of limited use; complete block of contrast material occurred, and the etiology of nerve root compression was not visualized. In isthmic spondylolisthesis, CT showed a fibrocartilaginous mass at the pars interarticularis, compressing the adjacent nerve root, as described by Gill et al. In degenerative spondylolisthesis, severe lateral recess stenosis occurred. CT provided diagnostic evidence of a tethered cord in dysplastic spondylolisthesis, if displaced bony fragments in the canal in traumatic spondylolisthesis, and spinal infiltration of neoplasms in the pathologic type. CT is important preoperatively in cases of severe spondylolisthesis and can be used to determine which warrant decompression in addition to fusion. These results also support the concept that neurologic findings associated with spondylolisthesis can be due to a multitude of anatomic abnormalities and should not be simply attributed to a herniated nucleus pulposis at the slip.
采用第四代计算机断层扫描(CT)诊断研究对连续31例腰椎滑脱患者的神经学改变进行调查或定位,并明确滑脱的解剖结构。18例患者在CT检查后接受了手术治疗,即脊柱融合术、减压术或两者皆有,平均随访时间为25个月。术后进行了10次CT研究以记录原始病变的矫正情况。15例患者存在与腰椎滑脱相关的客观神经功能缺损,其中13例CT显示了神经受压的具体部位。术前脊髓造影的作用有限;造影剂出现完全阻塞,神经根受压的病因无法显示。在峡部裂型腰椎滑脱中,CT显示关节突间部有纤维软骨肿块,压迫相邻神经根,如吉尔等人所述。在退变性腰椎滑脱中,出现了严重的侧隐窝狭窄。CT为发育不良型腰椎滑脱中的脊髓栓系、创伤性腰椎滑脱中椎管内移位的骨碎片以及病理类型中肿瘤的脊髓浸润提供了诊断依据。在严重腰椎滑脱病例中,CT在术前很重要,可用于确定哪些病例除融合外还需要减压。这些结果也支持这样一种观点,即与腰椎滑脱相关的神经学表现可能是由多种解剖异常引起的,不应简单地归因于滑脱处的髓核突出。