Hasin Y, Kriwisky M, Gotsman M S
Isr J Med Sci. 1982 Apr;18(4):499-502.
A patient with left ventricular aneurysm had recurrent ventricular tachycardia. His arrhythmia could be induced and interrupted by programmed ventricular stimulation. Quinidine and procainamide facilitated the induction of the tachycardia by widening the tachycardia induction zone. Ajmaline slowed the rate of the induced tachycardia and verapamil abolished the arrhythmia. The possible mechanism underlying these phenomena is discussed.
一名患有左心室动脉瘤的患者出现反复发作的室性心动过速。其心律失常可通过程控心室刺激诱发和终止。奎尼丁和普鲁卡因胺通过扩大心动过速诱发区促进了心动过速的诱发。阿吗灵减慢了诱发心动过速的速率，维拉帕米消除了心律失常。文中讨论了这些现象潜在的可能机制。
