3-甲基胆蒽和苏丹III对肝微粒体普萘洛尔N-去异丙基酶活性的诱导作用 - Suppr | 超能文献

Induction of hepatic microsomal propranolol N-desisopropylase activity by 3-methylcholanthrene and Sudan III.

作者信息

Fujita S, Usui T, Suzuki M, Hirasawa M, Mori Y, Naganuma H, Suzuki T

出版信息

Biochem Biophys Res Commun. 1982 Apr 14;105(3):1233-9. doi: 10.1016/0006-291x(82)91100-7.

DOI:10.1016/0006-291x(82)91100-7

Abstract

摘要

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

文档翻译

学术文献翻译模型，支持多种主流文档格式。

相似文献

1

Induction of hepatic microsomal propranolol N-desisopropylase activity by 3-methylcholanthrene and Sudan III.3-甲基胆蒽和苏丹III对肝微粒体普萘洛尔N-去异丙基酶活性的诱导作用

Biochem Biophys Res Commun. 1982 Apr 14;105(3):1233-9. doi: 10.1016/0006-291x(82)91100-7.

2

Species differences in stimulation of intestinal and hepatic microsomal mixed-function oxidase enzymes.肠道和肝脏微粒体混合功能氧化酶的刺激作用中的物种差异。

Biochem Pharmacol. 1980 Apr 15;29(8):1161-5. doi: 10.1016/0006-2952(80)90412-8.

3

Difference in the nature of induction of mixed-function oxidase systems of the rat liver among phenobarbital, DDT, 3-methylcholanthrene, and TCDD.苯巴比妥、滴滴涕、3-甲基胆蒽和四氯二苯并二恶英对大鼠肝脏混合功能氧化酶系统诱导性质的差异。

Toxicol Appl Pharmacol. 1981 Oct;61(1):109-18. doi: 10.1016/0041-008x(81)90012-0.

4

Cholestasis as an in vivo model for analysis of the induction of liver microsomal monooxygenases by sodium phenobarbital and 3-methylcholanthrene.胆汁淤积作为一种体内模型，用于分析苯巴比妥钠和3-甲基胆蒽对肝微粒体单加氧酶的诱导作用。

Biochem Pharmacol. 1979 May 1;28(9):1473-8. doi: 10.1016/0006-2952(79)90460-x.

5

Effects of 2,2'-dibromobiphenyl and 2,2',3,4,4',5,5'-heptabromobiphenyl on liver microsomal drug metabolizing enzymes.2,2'-二溴联苯和2,2',3,4,4',5,5'-七溴联苯对肝脏微粒体药物代谢酶的影响。

Toxicol Appl Pharmacol. 1979 Mar 30;48(1 Pt 1):73-86. doi: 10.1016/s0041-008x(79)80010-1.

6

Influence of 2,4-dichloro-6-phenoxyethyl-amine (DPEA) and -diethylaminoethyl diphenylpropylacetate (SKF-525A) on hepatic microsomal azoreductase activity from phenobarbital or 3-methylcholanthrene induced rats.2,4-二氯-6-苯氧基乙胺（DPEA）和二乙氨基乙基二苯基丙酯（SKF-525A）对苯巴比妥或3-甲基胆蒽诱导的大鼠肝微粒体偶氮还原酶活性的影响。

Biochem Pharmacol. 1972 Jan;21(1):69-75. doi: 10.1016/0006-2952(72)90251-1.

7

Induction of propranolol metabolism by the azo dye sudan III in rats.大鼠体内偶氮染料苏丹III对普萘洛尔代谢的诱导作用。

Biochem Pharmacol. 1992 Jun 9;43(11):2489-92. doi: 10.1016/0006-2952(92)90332-d.

8

Strain differences in the induction of soluble and microsomal enzymes by phenobarbital and 3-methylcholanthrene.苯巴比妥和3-甲基胆蒽对可溶性酶和微粒体酶诱导作用的品系差异。

Chem Biol Interact. 1974 May;8(5):297-302. doi: 10.1016/0009-2797(74)90008-8.

9

Comparison of hepatic microsomal enzyme induction by methadone, phenobarbital and 3-methylcholanthrene in the mouse.美沙酮、苯巴比妥和3-甲基胆蒽对小鼠肝微粒体酶诱导作用的比较。

Biochem Pharmacol. 1979 Dec 1;28(23):3476-82. doi: 10.1016/0006-2952(79)90091-1.

10

Effect of riboflavin deficiency on phenobarbital and 3-methylcholanthrene induction of microsomal drug-metabolizing enzymes of the rat.核黄素缺乏对大鼠微粒体药物代谢酶的苯巴比妥和3-甲基胆蒽诱导作用的影响。

Biochem Pharmacol. 1973 Oct 1;22(19):2365-73. doi: 10.1016/0006-2952(73)90338-9.

引用本文的文献

1

Selective effect of adjuvant arthritis on the disposition of propranolol enantiomers in rats detected using a stereospecific HPLC assay.使用立体特异性高效液相色谱法检测佐剂性关节炎对大鼠体内普萘洛尔对映体处置的选择性作用。

Pharm Res. 1993 Feb;10(2):294-9. doi: 10.1023/a:1018907431893.

2

Partial metabolic clearances as determinants of the oral bioavailability of propranolol.作为普萘洛尔口服生物利用度决定因素的部分代谢清除率。

Br J Clin Pharmacol. 1986 Sep;22(3):317-23. doi: 10.1111/j.1365-2125.1986.tb02893.x.