Inoue H, Takenaka K, Murayama M, Amano K, Uchiyama I, Kawahara T, Mashima S, Sakamoto T, Murao S
Jpn Heart J. 1982 May;23(3):279-92. doi: 10.1536/ihj.23.279.
Effects of cardiac size on surface potential magnitude were investigated by induction of acute changes in ventricular dimensions by rapid atrial pacing in 17 cases with ischemic heart disease (IHD) and in 15 cases without IHD. Left ventricular end-diastolic dimensions (LVDd), estimated by echocardiography, decreased along with certain changes in surface potential magnitude during atrial pacing in each case. A positive correlation between LVDd and the amplitudes of RV5, RX, and the maximum spatial QRS magnitude was observed in most of the cases and might be explained by changes in distance between the heart and the chest electrode and/or the Brody effect. However, an inverse correlation was observed in 4 of 17 cases with IHD, and could not be explained by these factors. Other factors, such as changes in the ventricular activation sequence, should be considered as the mechanism.
通过对17例缺血性心脏病(IHD)患者和15例非缺血性心脏病患者进行快速心房起搏,诱导心室尺寸急性变化,研究心脏大小对体表电位幅度的影响。通过超声心动图估计的左心室舒张末期内径(LVDd),在每例心房起搏过程中,随着体表电位幅度的某些变化而减小。在大多数病例中观察到LVDd与RV5、RX振幅以及最大空间QRS幅度之间呈正相关,这可能由心脏与胸部电极之间距离的变化和/或布罗迪效应来解释。然而,在17例IHD患者中有4例观察到负相关,这些因素无法解释这种负相关。其他因素,如心室激动顺序的变化,应被视为其机制。