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Behavioral changes in rats after chronic aluminum and parathyroid hormone administration.

作者信息

Commissaris R L, Cordon J J, Sprague S, Keiser J, Mayor G H, Rech R H

出版信息

Neurobehav Toxicol Teratol. 1982 May-Jun;4(3):403-10.

PMID:7099360
Abstract
摘要

相似文献

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Neurobehav Toxicol Teratol. 1982 May-Jun;4(3):403-10.
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Post-exposure self-recovery reverses oxidative stress, ameliorates pathology and neurotransmitters imbalance and rescues spatial memory after time-dependent aluminum exposure in rat brain.慢性铝暴露后，通过暴露后自身恢复可逆转氧化应激，改善病理学和神经递质失衡，并挽救大鼠大脑的空间记忆。
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2
Comparative Study of Time-Dependent Aluminum Exposure and Post-Exposure Recovery Shows Better Improvement in Synaptic Changes and Neuronal Pathology in Rat Brain After Short-Term Exposure.时间依赖性铝暴露与暴露后恢复的比较研究表明，短期暴露后大鼠大脑中突触变化和神经元病理学的改善更好。
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Expression of DnMTs and MBDs in AlCl-Induced Neurotoxicity Mouse Model.DnMTs 和 MBDs 在 AlCl 诱导的神经毒性小鼠模型中的表达。
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The effects of aluminum loading on selected tissue calcium and magnesium concentrations in rats.铝负荷对大鼠某些组织钙镁浓度的影响。
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5
Environmental effects of aluminium.铝的环境影响。
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6
Impairment of motor coordination in mice after ingestion of aluminum chloride.摄入氯化铝后小鼠运动协调性受损。
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Aluminum inhibition of human serum cholinesterase.
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The aluminum-induced increase in blood-brain barrier permeability to delta-sleep-inducing peptide occurs throughout the brain and is independent of phosphorus and acetylcholinesterase levels.铝诱导的血脑屏障对δ-睡眠诱导肽通透性的增加在全脑均有发生，且与磷和乙酰胆碱酯酶水平无关。
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