de Klerk A J, Jonck L M
S Afr Med J. 1982 Aug 21;62(9):276-81.
The management of a severed tendon within the digital sheath continues to present a formidable challenge. The major problem is one of excessive scar tissue formation. It was found experimentally in severed and partially divided tendons that the source of the blood supply, i.e. the vasculature in the tendon proper, will promote healing through the primary tissue (tenoblast) of the tendon itself, whereas the blood supply and granulation tissue from the damaged area of the flexor sheath leads to fibrous (scar) tissue formation. In this study of tendon repair special attention was paid to a suturing procedure which would have the least effect on the intrinsic blood supply to the severed ends. It was found that the basic requirement in successful tendon repair was to approximate and not to strangulate, and to minimize trauma to the flexor sheath which is the main source of scar tissue.
指腱鞘内肌腱断裂的处理仍然是一项艰巨的挑战。主要问题是过度形成瘢痕组织。通过实验发现，在完全断裂和部分断裂的肌腱中，血液供应的来源，即肌腱本身的脉管系统，将通过肌腱自身的原始组织（成腱细胞）促进愈合，而屈肌腱鞘受损区域的血液供应和肉芽组织会导致纤维（瘢痕）组织形成。在这项肌腱修复研究中，特别关注了一种对离断端固有血液供应影响最小的缝合方法。结果发现，成功进行肌腱修复的基本要求是对合而不是勒紧，并尽量减少对作为瘢痕组织主要来源的屈肌腱鞘的创伤。
