Nardoni A, Copetti R, Baldissera S, Busettini G, Cella R, Nador G
Minerva Med. 1982 Nov 10;73(43):3059-62.
The authors have investigated the changes of kalemia and kaluria levels in 40 patients suffering from delirium tremens, and have observed during the acute stage of the illness, a swift fall of kalemia level without an increase of the kaluric excretion. The kalemic values quickly rise up again at the end of the delirium.. The authors think that an adrenergic mechanism is responsible for this quick and transient hypokalemia. The significance of this adrenergic mechanism in the alcohol withdrawal syndrome is at present widely proved.
作者研究了40例震颤谵妄患者的血钾和尿钾水平变化，发现在疾病急性期血钾水平迅速下降，而尿钾排泄并未增加。谵妄结束时血钾值又迅速回升。作者认为，肾上腺素能机制是导致这种快速且短暂的低钾血症的原因。目前，这种肾上腺素能机制在酒精戒断综合征中的重要性已得到广泛证实。
