Sjaastad O, Russell D, Saunte C, Hørven I
Cephalalgia. 1982 Dec;2(4):211-4. doi: 10.1046/j.1468-2982.1982.0204211.x.
In four of the approximately 40 cases of chronic paroxysmal hemicrania (CPH) that are known so far, attacks that are similar to the spontaneous ones may be precipitated by head movements or pressure against certain points in the neck. Head flexion was used as the precipitation procedure in a 34-year-old female who was studied several times in the course of five years. Attacks occur within 5-40 sec, and the pain is preceded by tearing and conjunctival injection. External rubbing or external compression of the common and internal carotid arteries on the symptomatic side did not produce attacks. The combination of head flexion and external compression of the common or internal carotid arteries on the symptomatic side invariably produced an attack of usual severity and within the usual time. This investigation would seem to render unlikely the possibility of a primary vascular mediation of the signal from the neck to the ocular region. Sympathetic fibres are the likely mediators of the impulses from the neck to the ocular area.
在目前已知的约40例慢性阵发性偏侧头痛（CPH）病例中，有4例患者类似自发发作的头痛可因头部运动或按压颈部某些部位而诱发。一名34岁女性在五年内接受了多次研究，研究中采用头部前屈作为诱发程序。发作在5 - 40秒内出现，疼痛发作前有流泪和结膜充血。对症状侧颈总动脉和颈内动脉进行外部摩擦或外部压迫未诱发发作。症状侧头部前屈与颈总动脉或颈内动脉外部压迫相结合总是会在通常时间内引发通常严重程度的发作。这项研究似乎不太可能存在从颈部到眼部区域的信号由原发性血管介导的情况。交感神经纤维可能是从颈部到眼部区域冲动的介导者。
