[家族性红细胞ATP：丙酮酸磷酸转移酶缺乏症] - Suppr | 超能文献

[Familial erythrocyte ATP: pyruvate phosphotransferase deficiency].

作者信息

Torliński L, Karoń H, Czech A, Michalewska D, Duczmal B

出版信息

Acta Haematol Pol. 1982 Jul-Dec;13(3-4):157-63.

Abstract

摘要

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

文档翻译

学术文献翻译模型，支持多种主流文档格式。

相似文献

1

[Familial erythrocyte ATP: pyruvate phosphotransferase deficiency].[家族性红细胞ATP：丙酮酸磷酸转移酶缺乏症]

Acta Haematol Pol. 1982 Jul-Dec;13(3-4):157-63.

2

Pyruvate kinase deficiency.丙酮酸激酶缺乏症

Nihon Ketsueki Gakkai Zasshi. 1987 Dec;50(8):1445-52.

3

[Electrophoretic heterogeneity of pyruvate kinase in erythrocytes in patients with enzymopathies].[酶病患者红细胞中丙酮酸激酶的电泳异质性]

Acta Univ Palacki Olomuc Fac Med. 1985;111:467-73.

4

[Hereditary haemolytic anaemia due to pyruvate kinase deficiency. Prognosis of neonatal forms (author's transl)].丙酮酸激酶缺乏所致的遗传性溶血性贫血。新生儿型的预后（作者译）

Nouv Presse Med. 1982 Mar 13;11(12):917-9.

5

[Inherited hemolytic anemia due to pyruvate kinase deficiency: II. Density-layer centrifugation of erythrocytes (author's transl)].丙酮酸激酶缺乏所致的遗传性溶血性贫血：II. 红细胞的密度梯度离心法（作者译）

Med Klin. 1979 May 4;74(18):691-4.

6

Pyruvate kinase "Göttingen 1,2": congenital hemolytic anemia, evidence of double heterozygosity, and lack of enzyme cooperativity.丙酮酸激酶“哥廷根1,2”：先天性溶血性贫血、双重杂合性证据及酶协同性缺失

Hum Genet. 1982;60(4):381-6. doi: 10.1007/BF00569226.

7

[A patient with anemia and jaundice].[一名患有贫血和黄疸的患者]

Ned Tijdschr Geneeskd. 1976 Dec 25;120(52):2304-6.

8

[Physico-chemical and kinetic properties of pyruvate kinase in erythrocytes in enzymopathic hemolytic anemia].[酶病性溶血性贫血中红细胞丙酮酸激酶的物理化学及动力学特性]

Vnitr Lek. 1984 Jun;30(6):542-9.

9

[Chronic non-spherocytic hemolytic anemia caused by pyruvate kinase deficiency in a Costa Rican family carrying hemoglobin C disease].[哥斯达黎加一个携带血红蛋白C病的家庭中由丙酮酸激酶缺乏引起的慢性非球形细胞溶血性贫血]

Sangre (Barc). 1990 Apr;35(2):128-33.

10

[A case of familial pyruvate kinase deficiency associated with acute myelomonocytic leukemia].[一例与急性粒单核细胞白血病相关的家族性丙酮酸激酶缺乏症]

Rinsho Ketsueki. 1984 Jan;25(1):75-81.