Bhargava V, Goldberger A
J Electrocardiol. 1981;14(1):57-60. doi: 10.1016/s0022-0736(81)80029-5.
Previous studies based primarily on counts of QRS notches and slurs have suggested that myocardial infarction (MI) increases high frequency QRS components. However, this technique does not quantitatively measure total high frequency voltage. To further study this problem, a microprocessor ECG system was employed capable of averaging multiple QRS complexes to reduce noise. Power spectrum analysis of the QRS using a Fast Fourier Transform was then performed in lead II in ten normal subjects and ten patients with prior inferior MI. The infarct group showed a decrease in QRS potentials over a broad frequency range (7.81-304.7 Hz). Therefore, contrary to previous estimates, MI may attenuate high as well as low frequency QRS potentials as part of a general decrease in electromotive force.
以往主要基于QRS波切迹和顿挫计数的研究表明，心肌梗死（MI）会增加高频QRS成分。然而，该技术无法定量测量总的高频电压。为进一步研究此问题，采用了一种微处理器心电图系统，该系统能够对多个QRS波群进行平均以降低噪声。然后，对10名正常受试者和10名既往有下壁心肌梗死的患者在II导联进行了使用快速傅里叶变换的QRS波功率谱分析。梗死组在较宽的频率范围（7.81 - 304.7Hz）内QRS波电位降低。因此，与先前的估计相反，作为电动势普遍降低的一部分，心肌梗死可能会使高频和低频QRS波电位均减弱。
