Waxman M B, Staniloff H, Wald R W
J Electrocardiol. 1981;14(1):83-9. doi: 10.1016/s0022-0736(81)80034-9.
This is a report of a case of stable uninterrupted spontaneous ventricular tachycardia which could be temporarily terminated by a strong increase in vagal tone reflexly induced by phenylephrine, 0.2 mg I.V. Following its abolition with procaine amide, 100 mg I.V., ventricular tachycardia was found to be reinducible by deep inspiration for limited periods of time which varied directly with the magnitude of the inspiratory volume. This effect was enhanced by vagal inhibition (atropine, 0.6 mg I.V.) and attenuated or prevented by vagal stimulation (carotid sinus massage or edrophonium 10 mg I.V.). It was concluded that a deep inspiration can, under some circumstances, induce ventricular tachycardia by a mechanism of vagal withdrawal.
本文报告一例持续性稳定型自发性室性心动过速病例，静脉注射0.2mg去氧肾上腺素可通过反射性增强迷走神经张力暂时终止该心动过速。静脉注射100mg普鲁卡因酰胺消除室性心动过速后，发现深吸气在有限时间内可再次诱发室性心动过速，诱发时间与吸气量大小直接相关。迷走神经抑制（静脉注射0.6mg阿托品）可增强此效应，而迷走神经刺激（颈动脉窦按摩或静脉注射10mg依酚氯铵）可减弱或预防此效应。得出的结论是，在某些情况下，深吸气可通过迷走神经撤离机制诱发室性心动过速。
