Imre G, Bögi J
Klin Monbl Augenheilkd. 1980 Dec;177(6):798-801. doi: 10.1055/s-2008-1057730.
Corneal vascularization is attributable to the swelling and increased lactate concentration in the cornea, which are caused by damage to the oxidizing metabolism. Invasion of leukocytes stimulated by prostaglandins also promotes the increase in the lactate concentration. The following causal treatment is possible: 40% glucose to combat the swelling, 3% cystein to improve the oxidizing metabolism and counteract the increased lactate concentration, and 0.5% indomethacin suspension to prevent the formation of prostaglandins and hence the invasion of leukozytes.
角膜血管化归因于角膜肿胀和乳酸浓度增加,这是由氧化代谢受损引起的。前列腺素刺激白细胞的侵入也会促使乳酸浓度升高。以下是可行的病因治疗方法:用40%的葡萄糖对抗肿胀,用3%的半胱氨酸改善氧化代谢并抵消升高的乳酸浓度,用0.5%的吲哚美辛混悬液防止前列腺素的形成,从而防止白细胞的侵入。