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Characterization and properties of repair of potentially lethal damage as measured with the help of beta-arabinofuranosyladenine in plateau-phase EAT cells.

作者信息

Iliakis G

出版信息

Radiat Res. 1981 Apr;86(1):77-90.

PMID:7220806
Abstract
摘要

相似文献

1
Characterization and properties of repair of potentially lethal damage as measured with the help of beta-arabinofuranosyladenine in plateau-phase EAT cells.用β - 阿拉伯呋喃糖基腺嘌呤测定高原期EAT细胞潜在致死性损伤修复的特征及性质。
Radiat Res. 1981 Apr;86(1):77-90.
2
Effects of beta-arabinofuranosyladenine on the growth and repair of potentially lethal damage in Ehrilch ascites tumor cells.β-阿拉伯呋喃糖基腺嘌呤对艾氏腹水瘤细胞潜在致死性损伤的生长和修复的影响。
Radiat Res. 1980 Sep;83(3):537-52.
3
Conditions supporting repair of potentially lethal damage cause a significant reduction of ultraviolet light-induced division delay in synchronized and plateau-phase Ehrlich ascites tumor cells.支持潜在致死性损伤修复的条件会使同步化和平台期艾氏腹水肿瘤细胞中紫外线诱导的分裂延迟显著降低。
Radiat Res. 1982 Sep;91(3):483-506.
4
Repair of potentially lethal damage in unfed plateau phase cultures of Ehrlich ascites tumour cells. II. Monolayer cultures.
Int J Radiat Biol Relat Stud Phys Chem Med. 1980 Jun;37(6):591-600.
5
Evidence that repair and expression of potentially lethal damage cause the variations in cell survival after X irradiation observed through the cell cycle in Ehrlich ascites tumor cells.通过艾氏腹水瘤细胞的细胞周期观察到，潜在致死性损伤的修复和表达会导致X射线照射后细胞存活的变化，这一证据表明了上述观点。
Radiat Res. 1983 Jul;95(1):87-107.
6
Modifications in repair and expression of potentially lethal damage (alpha-PLD) as measured by delayed plating or treatment with beta-araA in plateau-phase Ehrlich ascites tumor cells after exposure to charged particles of various specific energies.在暴露于各种特定能量的带电粒子后，通过延迟铺板或用β-阿拉伯糖胞苷处理处于平台期的艾氏腹水瘤细胞，来测量潜在致死性损伤（α-PLD）的修复和表达的变化。
Radiat Res. 1987 Jul;111(1):26-46.
7
Effect of arabinofuranosyladenine on radiation-induced chromosome damage in plateau-phase CHO cells measured by premature chromosome condensation: implications for repair and fixation of alpha-PLD.通过早熟染色体凝集测定阿拉伯糖基腺嘌呤对处于平台期的中国仓鼠卵巢（CHO）细胞辐射诱导染色体损伤的影响：对α粒子诱发的潜在致死性损伤修复和固定的意义
Radiat Res. 1988 May;114(2):361-78.
8
Effects of ara A and fresh medium on chromosome damage and DNA double-strand break repair in X-irradiated stationary cells.阿糖腺苷和新鲜培养基对X射线照射的静止细胞中染色体损伤和DNA双链断裂修复的影响。
Br J Cancer Suppl. 1984;6:61-5.
9
Effects of the nucleoside analogues alpha-ara A, beta-ara A and beta-ara C on cell growth and repair of both potentially lethal damage and DNA double strand breaks in mammalian cells in culture.核苷类似物α-阿糖腺苷、β-阿糖腺苷和β-阿糖胞苷对培养的哺乳动物细胞的细胞生长、潜在致死性损伤修复及DNA双链断裂的影响。
Anticancer Res. 1983 Mar-Apr;3(2):143-9.
10
Inhibitors of repair of potentially lethal damage and DNA polymerases also influence the recovery of potentially neoplastic transforming damage in C3H10T-1/2 cells.潜在致死性损伤修复抑制剂和DNA聚合酶也会影响C3H10T-1/2细胞中潜在致癌转化损伤的恢复。
Radiat Res. 1990 Jul;123(1):49-54.

引用本文的文献

1
The importance of G1/S-border and mitosis in the fixation of potentially lethal damage.
Radiat Environ Biophys. 1983;22(3):201-7. doi: 10.1007/BF01323709.
2
The influence of PLD repair on the induction frequency of 6-thioguanine resistant mutants in Ehrlich ascites tumour cells after exposure to X-rays and ultraviolet light.PLD修复对艾氏腹水瘤细胞经X射线和紫外线照射后6-硫鸟嘌呤抗性突变体诱导频率的影响。
Br J Cancer Suppl. 1984;6:113-7.
3
Evidence for the induction of two types of potentially lethal damage after exposure of plateau phase Chinese hamster V79 cells to gamma-rays.高原期中国仓鼠V79细胞经γ射线照射后诱导产生两种潜在致死性损伤的证据。
Radiat Environ Biophys. 1985;24(3):185-202. doi: 10.1007/BF01209522.
4
Effects of adenosine deaminase inhibitor 2'-deoxycoformycin on the repair and expression of potentially lethal damage sensitive to beta-araA.腺苷脱氨酶抑制剂2'-脱氧助间型霉素对β-阿拉伯糖胞苷敏感的潜在致死性损伤修复及表达的影响
Radiat Environ Biophys. 1985;24(2):81-8. doi: 10.1007/BF01229813.
5
A cell survival model with saturable repair after irradiation.一种照射后具有饱和修复的细胞存活模型。
Radiat Environ Biophys. 1987;26(1):63-79. doi: 10.1007/BF01211365.
6
Repair and fixation of potentially lethal damage (PLD) as demonstrated by delayed plating or incubation with araA in contact inhibited refed plateau-phase C3H mouse embryo 10 T1/2 cells grown in the presence of BrdUrd.在BrdUrd存在的情况下培养的接触抑制的再喂食平台期C3H小鼠胚胎10 T1/2细胞中，通过延迟铺板或与阿糖腺苷一起孵育所证明的潜在致死性损伤（PLD）的修复和固定。
Radiat Environ Biophys. 1987;26(1):47-62. doi: 10.1007/BF01211364.
7
Evidence for induction and repair of potentially lethal damage in plateau-phase V79 cells after exposure to adriamycin. The importance of removal of adriamycin released from the cells during the post-treatment incubation period.阿霉素处理后处于平台期的V79细胞中潜在致死性损伤的诱导与修复证据。后处理孵育期细胞释放的阿霉素清除的重要性。
Br J Cancer. 1987 Apr;55(4):381-4. doi: 10.1038/bjc.1987.76.
8
Differences in inhibition by beta-arabinofuranosyladenine (araA) of radiation induced DNA damage repair in exponentially growing and plateau-phase CHO-cells.β-阿拉伯呋喃糖基腺嘌呤（araA）对指数生长期和平稳期CHO细胞辐射诱导的DNA损伤修复抑制作用的差异。
Radiat Environ Biophys. 1988;27(4):295-305. doi: 10.1007/BF01209758.