Saad W A, Camargo L A, Silva-Netto C R, Antunes-Rodrigues J, Covian M R
Brain Res Bull. 1981 Mar;6(3):197-202. doi: 10.1016/s0361-9230(81)80047-0.
Sodium chloride intake was studied in rats submitted to different neurosurgical procedures. Intake decreased in animals submitted to bilateral destruction of the basolateral amygdaloid complex, and increased after the same animals were submitted to destruction of the anterior lateral hypothalamus, a procedure which is known to cause increased intake in intact rats. In the reverse experiment, where the anterior lateral hypothalamus was destroyed before the basolateral amygdaloid complex, the effect of increased sodium chloride intake induced by destruction of the hypothalamus overcame the decreased expected upon destruction of the amygdaloid complex. These results permit us to conclude that the hypothalamic areas which inhibit sodium chloride intake predominate over the stimulating areas of the amygdaloid complex in the control of sodium chloride intake.
对接受不同神经外科手术的大鼠的氯化钠摄入量进行了研究。双侧基底外侧杏仁核复合体损毁的动物摄入量减少，而在同一批动物接受下丘脑外侧前部损毁后摄入量增加，已知该手术会使正常大鼠的摄入量增加。在反向实验中，先损毁下丘脑外侧前部，再损毁基底外侧杏仁核复合体，下丘脑损毁引起的氯化钠摄入量增加的效应超过了杏仁核复合体损毁预期会出现的摄入量减少。这些结果使我们能够得出结论，在控制氯化钠摄入量方面，抑制氯化钠摄入的下丘脑区域比杏仁核复合体的刺激区域占主导地位。
