Perenin M T, Ruel J, Hécaen H
Cortex. 1980 Dec;16(4):605-12. doi: 10.1016/s0010-9452(80)80007-4.
A case of cortical blindness resulting from a dense ischemic lesion of both calcarine cortices (as seen on CAT Scan) was studied up till the seventh month after the initial stroke. By using mainly forced-choice procedures, similar to those previously used for testing hemianopic subjects, we were able to demonstrate the reappearance of some visual capacities, even though the patient still behaved as if completely blind in everyday life and the lesion remained as it was first seen. First, an ability to detect moving stimuli reappeared then bright flashes could be detected. At last, the patient could localize flickering spots approximately, by pointing with his hand, despite the fact that he did not really see them. As was hypothesized in cases of unilateral occipital lesions, such residual vision would likely to be subserved by extrageniculostriate pathways.
有一例因双侧距状皮质严重缺血性病变(计算机断层扫描所见)导致的皮质盲患者,在首次中风后直至第七个月都在接受研究。主要采用类似于先前用于测试偏盲患者的迫选程序,我们能够证明一些视觉能力重新出现,尽管患者在日常生活中仍表现得好像完全失明,且病变仍与最初所见相同。首先,检测移动刺激的能力重新出现,然后能够检测到明亮的闪光。最后,尽管患者并未真正看见,但他能用手指大致定位闪烁的光点。正如单侧枕叶病变病例中所假设的那样,这种残余视力可能由膝状体纹外通路提供支持。