Topilow H W
Ann Ophthalmol. 1981 Feb;13(2):221-5.
Discrete lesions interrupting the fiber tracts that connect the pontine center for conjugate horizontal gaze and the ipsilateral abducens nucleus create the syndrome of Lutz's posterior internuclear ophthalmoplegia. In this syndrome the lateral rectus on the side of the lesion fails to abduct the eye normally on attempted horizontal gaze to the same side. Adduction of the contralateral eye is normal. This syndrome may be differentiated from the more common abducens nerve palsy by noting the orthotropic position of the eyes in primary position, the absence of primary and secondary deviations with alternate fixation, and the absence of diplopia. A case of this syndrome, presumably due to central nervous system vasculitis, is reported, and related human and animal studies are reviewed.
中断连接脑桥共轭水平凝视中枢与同侧展神经核的纤维束的离散性病变会导致卢茨后核间性眼肌麻痹综合征。在该综合征中，病变侧的外直肌在试图向同侧水平凝视时不能正常外展眼球。对侧眼的内收正常。通过注意双眼在初始位置的正视位、交替注视时无原发性和继发性斜视以及无复视，可将该综合征与更常见的展神经麻痹相鉴别。本文报告了一例推测由中枢神经系统血管炎引起的该综合征病例，并对相关的人体和动物研究进行了综述。
