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Comparison of the effects of griseofulvin and 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine on ferrochelatase activity in chick embryo liver.

作者信息

Cole S P, Zelt D T, Marks G S

出版信息

Mol Pharmacol. 1981 May;19(3):477-80.

PMID:7266472
Abstract
摘要

相似文献

1
Comparison of the effects of griseofulvin and 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine on ferrochelatase activity in chick embryo liver.灰黄霉素与3,5 - 二乙氧基羰基 - 1,4 - 二氢 - 2,4,6 - 三甲基吡啶对鸡胚肝脏中铁螯合酶活性影响的比较
Mol Pharmacol. 1981 May;19(3):477-80.
2
Ferrochelatase-inhibitory and porphyrin-inducing properties of 3,5-diethoxycarbonyl-1, 4-dihydro-2,4,6-trimethylpyridine and its analogues in chick embryo liver cells.3,5-二乙氧基羰基-1,4-二氢-2,4,6-三甲基吡啶及其类似物在鸡胚肝细胞中的亚铁螯合酶抑制和卟啉诱导特性
Mol Pharmacol. 1981 Sep;20(2):395-403.
3
Drug-induced porphyrin biosynthesis--XIX. Potentiation of the porphyrin-inducing effects of SKF 525-A in the chick embryo liver by 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine, an antihibitor of ferrochelatase.药物诱导的卟啉生物合成——十九。3,5 - 二乙氧羰基 - 1,4 - 二氢 - 2,4,6 - 三甲基吡啶（一种亚铁螯合酶抑制剂）对SKF 525 - A在鸡胚肝脏中诱导卟啉作用的增强。
Biochem Pharmacol. 1979 Dec 15;28(24):0533-8. doi: 10.1016/0006-2952(79)90397-6.
4
Ferrochelatase-inhibitory activity and N-alkylprotoporphyrin formation with analogues of 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine (DDC) containing extended 4-alkyl groups: implications for the active site of ferrochelatase.含延长4-烷基的3,5-二乙氧羰基-1,4-二氢-2,4,6-三甲基吡啶（DDC）类似物的亚铁螯合酶抑制活性及N-烷基原卟啉的形成：对亚铁螯合酶活性位点的意义
Mol Pharmacol. 1986 Oct;30(4):352-7.
5
Suicidal destruction of cytochrome P-450 and reduction of ferrochelatase activity by 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine and its analogues in chick embryo liver cells.3,5 - 二乙氧基羰基 - 1,4 - 二氢 - 2,4,6 - 三甲基吡啶及其类似物对鸡胚肝细胞中细胞色素P - 450的自杀性破坏及亚铁螯合酶活性的降低
Mol Pharmacol. 1985 Apr;27(4):459-65.
6
Effects of porphyrin-inducing drugs on ferrochelatase activity in isolated mouse hepatocytes.卟啉诱导药物对分离的小鼠肝细胞中铁螯合酶活性的影响。
Can J Physiol Pharmacol. 1981 Nov;59(11):1155-8. doi: 10.1139/y81-178.
7
Comparison of the effects of 3-ethoxycarbonyl-1,4-dihydro-2,4-dimethylpyridine and 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine on ferrochelatase activity and heme biosynthesis in chick embryo liver cells in culture.
Can J Physiol Pharmacol. 1986 Apr;64(4):483-6. doi: 10.1139/y86-078.
8
Inhibition of ferrochelatase by N-methylprotoporphyrin IX is not accompanied by delta-aminolevulinic acid synthetase induction in chick embryo liver cell culture.在鸡胚肝细胞培养中，N-甲基原卟啉IX对亚铁螯合酶的抑制作用并未伴随δ-氨基乙酰丙酸合成酶的诱导。
Can J Physiol Pharmacol. 1982 Feb;60(2):212-5. doi: 10.1139/y82-034.
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Porphyrinogenic activity and ferrochelatase-inhibitory activity of sydnones in chick embryo liver cells.
FEBS Lett. 1986 Mar 3;197(1-2):17-20. doi: 10.1016/0014-5793(86)80289-7.
10
Isolation of an N-alkylprotoporphyrin IX from chick embryo livers following the administration of 3,5-diethoxycarbonyl-1,4-dihydro-4-ethyl-2,6-dimethylpyridine.
Can J Physiol Pharmacol. 1987 Jul;65(7):1500-2. doi: 10.1139/y87-235.

引用本文的文献

1
Protoporphyrin IX: the Good, the Bad, and the Ugly.原卟啉IX：好坏参半。
J Pharmacol Exp Ther. 2016 Feb;356(2):267-75. doi: 10.1124/jpet.115.228130. Epub 2015 Nov 20.
2
Ferrochelatase and N-alkylated porphyrins.亚铁螯合酶与N-烷基化卟啉
Mol Cell Biochem. 1984 Sep;64(2):127-37. doi: 10.1007/BF00224769.
3
Inhibition of ferrochelatase and accumulation of porphyrins in mouse hepatocyte cultures exposed to porphyrinogenic chemicals.
Arch Toxicol. 1992;66(3):175-81. doi: 10.1007/BF01974011.