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[实验性心绞痛时电刺激颈动脉窦神经期间犬心肌和颈动脉体的功能形态学]

[Functional morphology of the myocardium and carotid paraganglion of dogs during electrical stimulation of the carotid sinus nerve in experimental stenocardia].

作者信息

Denisov I N, Kvetnoĭ I M

出版信息

Arkh Patol. 1981;43(7):55-8.

PMID:7271507
Abstract

Histochemical methods demonstrated the recovery of the glycogen content, the activity of succinate dehydrogenase and alpha-glycerophosphate dehydrogenase as well as increased fluorescence of catecholaminergic nerves in short-term ischemized myocardium (experimental angina pectoris) after stimulation of the carotid sinus nerve. At the same time, a decrease in the number of cells containing catecholamines in the carotid paraganglion was observed which might indicate their discharge into the blood. Normalization of the histochemical values in ischemized myocardium after stimulation of the carotid sinus nerve allows this method to recommend for investigations in clinical practice.

摘要

组织化学方法显示,在刺激颈动脉窦神经后,短期缺血心肌(实验性心绞痛)中糖原含量、琥珀酸脱氢酶和α-甘油磷酸脱氢酶的活性得以恢复,同时儿茶酚胺能神经的荧光增强。与此同时,观察到颈动脉旁神经节中含儿茶酚胺的细胞数量减少,这可能表明它们释放到了血液中。刺激颈动脉窦神经后缺血心肌组织化学值的正常化使得该方法可推荐用于临床实践研究。

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