Walter H, Schliwa R, Lankenau H, Singh I P, Bhasin M K, Veerraju P, Goud J D, Nemeskeri J
Acta Anthropogenet. 1981;5(2):137-46.
Population samples from Hungary and India have been typed for beta 2-glycoprotein I concentrations. Whereas the Hungarian sample is in fairly good accord with the genetic model set up by Cleve2-beta 2-glycoprotein I concentrations are controlled by two autosomal codominant alleles BgN and BgD-the Indian samples do not fit this model. Thus the Indian data favour the assumption of a more complex genetic mechanism controlling the serum concentration of this protein.
已对来自匈牙利和印度的人群样本进行β2-糖蛋白I浓度分型。匈牙利样本与Cleve建立的遗传模型相当吻合——β2-糖蛋白I浓度由两个常染色体共显性等位基因BgN和BgD控制——而印度样本并不符合该模型。因此，印度的数据支持存在一种更复杂的遗传机制控制该蛋白血清浓度这一假设。
