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On the relationship between kainic acid-induced epileptiform activity and hippocampal neuronal damage.

作者信息

Sloviter R S, Damiano B P

出版信息

Neuropharmacology. 1981 Nov;20(11):1003-11. doi: 10.1016/0028-3908(81)90088-5.

DOI:10.1016/0028-3908(81)90088-5
PMID:7322276
Abstract
摘要

相似文献

1
On the relationship between kainic acid-induced epileptiform activity and hippocampal neuronal damage.
Neuropharmacology. 1981 Nov;20(11):1003-11. doi: 10.1016/0028-3908(81)90088-5.
2
Do kainate-lesioned hippocampi become epileptogenic?海人酸损伤的海马体是否会变得具有致痫性？
Brain Res. 1985 Mar 11;329(1-2):309-13. doi: 10.1016/0006-8993(85)90540-2.
3
Sustained electrical stimulation of the perforant path duplicates kainate-induced electrophysiological effects and hippocampal damage in rats.对大鼠的穿通通路进行持续电刺激可重现红藻氨酸诱导的电生理效应及海马损伤。
Neurosci Lett. 1981 Jul 17;24(3):279-84. doi: 10.1016/0304-3940(81)90171-3.
4
A role for sodium and chloride in kainic acid-induced beading of inhibitory interneuron dendrites.钠和氯在海藻酸诱导的抑制性中间神经元树突串珠形成中的作用。
Neuroscience. 2000;101(2):337-48. doi: 10.1016/s0306-4522(00)00384-5.
5
Hyperexcitability and cell loss in kainate-treated hippocampal slice cultures.
Neuroreport. 1993 Oct 25;5(1):90-2. doi: 10.1097/00001756-199310000-00025.
6
Intraventricular kainic acid preferentially destroys hippocampal pyramidal cells.
Nature. 1978 Feb 16;271(5646):676-7. doi: 10.1038/271676a0.
7
Focal inhibitory interneuron loss and principal cell hyperexcitability in the rat hippocampus after microinjection of a neurotoxic conjugate of saporin and a peptidase-resistant analog of Substance P.在向大鼠海马体微量注射皂草素与P物质的抗肽酶类似物的神经毒性偶联物后，局部抑制性中间神经元丧失及主细胞过度兴奋。
J Comp Neurol. 2001 Jul 23;436(2):127-52.
8
Intrahippocampal kainic acid, seizures and local neuronal degeneration: relationships assessed in unanesthetized rats.海马内注射 kainic 酸、癫痫发作与局部神经元变性：在未麻醉大鼠中评估的关系
Neuroscience. 1982 Oct;7(10):2525-36. doi: 10.1016/0306-4522(82)90212-3.
9
The effect of kainic acid on excitatory synaptic activity in the rat hippocampal slice preparation.红藻氨酸对大鼠海马脑片制备中兴奋性突触活动的影响。
Neurosci Lett. 1981 Nov 18;27(1):31-6. doi: 10.1016/0304-3940(81)90201-9.
10
Short-term effects of kainic acid on CA1 hippocampal interneurons differentially vulnerable to excitotoxicity.海藻酸对海马CA1区不同易受兴奋毒性影响的中间神经元的短期作用。
Epilepsia. 2005 Jun;46(6):837-48. doi: 10.1111/j.1528-1167.2005.21404.x.

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Minimal latency to hippocampal epileptogenesis and clinical epilepsy after perforant pathway stimulation-induced status epilepticus in awake rats.清醒大鼠穿通通路刺激诱导癫痫持续状态后海马癫痫发生和临床癫痫的最短潜伏期。
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PLoS One. 2007 Oct 31;2(10):e1105. doi: 10.1371/journal.pone.0001105.
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Synaptic kainate currents reset interneuron firing phase.突触海人藻酸电流重置中间神经元放电相位。
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Kainate receptors regulate unitary IPSCs elicited in pyramidal cells by fast-spiking interneurons in the neocortex.海人酸受体调节新皮质中快速放电中间神经元在锥体细胞中引发的单位抑制性突触后电流。
J Neurosci. 2001 May 1;21(9):2992-9. doi: 10.1523/JNEUROSCI.21-09-02992.2001.
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Kainate receptor-mediated responses in the CA1 field of wild-type and GluR6-deficient mice.野生型和GluR6基因缺陷型小鼠CA1区中红藻氨酸受体介导的反应。
J Neurosci. 1999 Jan 15;19(2):653-63. doi: 10.1523/JNEUROSCI.19-02-00653.1999.