Tajiri J, Nakayama M, Sato T, Isozaki S, Uchino K
Jpn J Med. 1981 Jul;20(3):216-21. doi: 10.2169/internalmedicine1962.20.216.
Batter's syndrome characteristically exhibits the constellation of hypokalemic alkalosis, normotensive hyperreninism, hyperalodosteronism, hyporesponsiveness to pressor agent and juxtaglomerular cell hyperplasia. Recently, metabolic mimicry of Batter's syndrome by vomiting, diarrhea, laxatives and diuretics abuse has been reported. We had a 30 year-old female patient who developed so-called pseudo-Bartter's syndrome as the result of surreptitious self-administration of furosemide for about six years. In this case, calcification of bilateral renal medulla was demonstrated. Such adverse reaction has not been reported to date. Moreover, a total 14 cases of pseudo-Bartter's syndrome reported in Japanese literature is reviewed.
巴特综合征的典型表现为低钾性碱中毒、血压正常的高肾素血症、高醛固酮血症、对升压药反应低下以及肾小球旁细胞增生。近来，有报道称呕吐、腹泻、滥用泻药和利尿剂可导致巴特综合征的代谢性模仿。我们有一位30岁女性患者，因秘密自行服用呋塞米约六年而患上所谓的假性巴特综合征。在该病例中，双侧肾髓质出现钙化。这种不良反应迄今尚未见报道。此外，还对日本文献中报道的总共14例假性巴特综合征病例进行了综述。
