Perez J E, Cintrón G, Gonzalez M, Hernández E, Linares E, Aranda J M
Chest. 1980 Feb;77(2):194-7. doi: 10.1378/chest.77.2.194.
Thirteen patients underwent right cardiac catheterization during the first 24 hours after the onset of symptoms of acute myocardial infarction. All had normal (less than 12 mm Hg) pulmonary arterial diastolic pressure or normal mean pulmonary wedge pressure (7.6 +/- 0.6 mm Hg). The patients did isometric forearm contraction to a measured level of 100 mm Hg. All patients had elevation of systemic systolic blood pressure, with a mean rise of 14.2 +/- 2.9 mm Hg; heart rate rose by 12 +/- 2 beats per minute. When compared to a control group, patients with acute myocardial infarction had a significant (P less than 0.005) elevation of 5.4 +/- 1.3 mm Hg in the pulmonary arterial diastolic or pulmonary wedge pressure. This may be a result of either increased left ventricular stiffness or decreased myocardial functional reserve. In the setting of acute myocardial infarction, patients with normal left ventricular filling pressures have abnormal ventricular performance. Isometric effort is poorly tolerated and should be avoided.
13例患者在急性心肌梗死症状发作后的最初24小时内接受了右心导管检查。所有患者的肺动脉舒张压均正常（低于12 mmHg）或平均肺楔压正常（7.6±0.6 mmHg）。患者进行等长前臂收缩至测量水平100 mmHg。所有患者的体循环收缩压均升高，平均升高14.2±2.9 mmHg；心率每分钟增加12±2次。与对照组相比，急性心肌梗死患者的肺动脉舒张压或肺楔压显著升高（P<0.005）5.4±1.3 mmHg。这可能是左心室僵硬度增加或心肌功能储备降低的结果。在急性心肌梗死的情况下，左心室充盈压正常的患者心室功能异常。等长运动耐受性差，应避免。
