一名先天性α2-纤溶酶抑制剂缺乏患者的纤溶状态 - Suppr

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超能文献

Fibrinolytic states in a patient with congenital deficiency of alpha 2-plasmin inhibitor.

作者信息

Aoki N, Sakata Y, Matsuda M, Tateno K

出版信息

Blood. 1980 Mar;55(3):483-8.

PMID:7357079

Abstract

摘要

相似文献

Fibrinolytic states in a patient with congenital deficiency of alpha 2-plasmin inhibitor.一名先天性α2-纤溶酶抑制剂缺乏患者的纤溶状态

Blood. 1980 Mar;55(3):483-8.

Differences between neonates and adults in the urokinase-plasminogen activator (u-PA) pathway of the fibrinolytic system.新生儿与成人在纤溶系统的尿激酶-纤溶酶原激活物（u-PA）途径中的差异。

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Secondary-site binding of Glu-plasmin, Lys-plasmin and miniplasmin to fibrin.谷氨酸纤溶酶、赖氨酸纤溶酶和微型纤溶酶与纤维蛋白的次要位点结合。

Biochem J. 1981 Sep 1;197(3):619-28. doi: 10.1042/bj1970619.

Increased urokinase and consumption of α -antiplasmin as an explanation for the loss of benefit of tranexamic acid after treatment delay.纤溶酶原和 α2-抗纤溶酶的消耗增加可解释为何氨甲环酸治疗延迟后获益丧失。

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The fibrinolytic system in man.人类的纤维蛋白溶解系统。

Crit Rev Oncol Hematol. 1986;4(3):249-301. doi: 10.1016/s1040-8428(86)80014-2.

Effects of pro-urokinase and urokinase on the fibrinolytic system in man.

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Effects of kringles derived from human plasminogen on fibrinolysis in vitro.源自人纤溶酶原的kringle结构域在体外对纤维蛋白溶解的影响。

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Plasma tissue-type plasminogen activator increases fibrinolytic activity of exogenous urokinase-type plasminogen activator.血浆组织型纤溶酶原激活物可增强外源性尿激酶型纤溶酶原激活物的纤溶活性。

Blood Coagul Fibrinolysis. 2012 Dec;23(8):729-33. doi: 10.1097/MBC.0b013e32835897d5.

Inherited disorders of the fibrinolytic pathway.纤维蛋白溶解途径的遗传性疾病。

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Comparison of the relative fibrinogenolytic, fibrinolytic and thrombolytic properties of tissue plasminogen activator and urokinase in vitro.组织型纤溶酶原激活剂与尿激酶在体外的相对纤维蛋白溶解、纤溶及溶栓特性比较

Thromb Haemost. 1981 Jun 30;45(3):225-9.

引用本文的文献

Significance of cross-linking of alpha 2-plasmin inhibitor to fibrin in inhibition of fibrinolysis and in hemostasis.α2-纤溶酶抑制剂与纤维蛋白交联在抑制纤维蛋白溶解和止血中的意义。

J Clin Invest. 1982 Mar;69(3):536-42. doi: 10.1172/jci110479.

Rapid purification of a high-affinity plasminogen activator from human blood plasma by specific adsorption on fibrin/Celite.通过在纤维蛋白/硅藻土上特异性吸附从人血浆中快速纯化高亲和力纤溶酶原激活剂。

Proc Natl Acad Sci U S A. 1981 Jul;78(7):4265-9. doi: 10.1073/pnas.78.7.4265.

Alpha2-plasmin inhibitor and alpha2-macroglobulin-plasmin complexes in plasma. Quantitation by an enzyme-linked differential antibody immunosorbent assay.血浆中的α2-纤溶酶抑制剂和α2-巨球蛋白-纤溶酶复合物。通过酶联差异抗体免疫吸附测定法定量。

J Clin Invest. 1981 Jul;68(1):46-55. doi: 10.1172/jci110253.

Organization of the human alpha 2-plasmin inhibitor gene.人α2-纤溶酶抑制剂基因的组织

Proc Natl Acad Sci U S A. 1988 Sep;85(18):6836-40. doi: 10.1073/pnas.85.18.6836.

Localization of the binding site of tissue-type plasminogen activator to fibrin.组织型纤溶酶原激活剂与纤维蛋白结合位点的定位

J Clin Invest. 1986 Jul;78(1):163-9. doi: 10.1172/JCI112546.

Molecular basis for congenital deficiency of alpha 2-plasmin inhibitor. A frameshift mutation leading to elongation of the deduced amino acid sequence.α2-纤溶酶抑制剂先天性缺乏的分子基础。一种导致推导的氨基酸序列延长的移码突变。

J Clin Invest. 1989 May;83(5):1598-604. doi: 10.1172/JCI114057.

The mechanism of activation of plasminogen at the fibrin surface by tissue-type plasminogen activator in a plasma milieu in vitro. Role of alpha 2-antiplasmin.体外血浆环境中组织型纤溶酶原激活物在纤维蛋白表面激活纤溶酶原的机制。α2-抗纤溶酶的作用。

Biochem J. 1990 Oct 1;271(1):51-7. doi: 10.1042/bj2710051.

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