Wagner G, Levin R J
Acta Pharmacol Toxicol (Copenh). 1980 May;46(5):321-5. doi: 10.1111/j.1600-0773.1980.tb02461.x.
No experimental data on the regulatory mechanism of the change in vaginal blood flow occurring at sexual arousal exist. Six women were in a controlled laboratory study given atropine 0.035 mg/kg intravenously. The basal vaginal blood flow was recorded by a heat probe kept at set temperature on the vaginal wall. During sexual stimulation the flow was increased as in women when no drugs are applied and orgasm was unaffected as well. The neurotransmitter has been supposed to be acetylcholine but the present experiments suggest that it is not an atropine sensitive traditional muscarinic transmission. Methylatropine was given in five subjects and neither in these cases any effect on the vaginal vascular response was observed.
目前尚无关于性唤起时阴道血流变化调节机制的实验数据。在一项对照实验室研究中，对6名女性静脉注射0.035mg/kg阿托品。通过置于阴道壁设定温度的热探头记录基础阴道血流。在性刺激期间，血流如未用药的女性一样增加，且性高潮也未受影响。曾推测神经递质为乙酰胆碱，但目前的实验表明，这不是对阿托品敏感的传统毒蕈碱传递。对5名受试者给予甲基阿托品，在这些情况下均未观察到对阴道血管反应有任何影响。
