高甘油三酯血症极低密度脂蛋白对培养的牛主动脉内皮细胞3-羟基-3-甲基戊二酰辅酶A还原酶活性及细胞活力的异常影响。 - Suppr

Suppr

超能文献

Abnormal effects of hypertriacylglycerolemic very low-density lipoproteins on 3-hydroxy-3-methylglutaryl-CoA reductase activity and viability of cultured bovine aortic endothelial cells.

作者信息

Gianturco S H, Eskin S G, Navarro L T, Lahart C J, Smith L C, Gotto A M

出版信息

Biochim Biophys Acta. 1980 Apr 18;618(1):143-52. doi: 10.1016/0005-2760(80)90061-2.

DOI:10.1016/0005-2760(80)90061-2

PMID:7378428

Abstract

摘要

相似文献

Abnormal effects of hypertriacylglycerolemic very low-density lipoproteins on 3-hydroxy-3-methylglutaryl-CoA reductase activity and viability of cultured bovine aortic endothelial cells.高甘油三酯血症极低密度脂蛋白对培养的牛主动脉内皮细胞3-羟基-3-甲基戊二酰辅酶A还原酶活性及细胞活力的异常影响。

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Abnormal suppression of 3-hydroxy-3-methylglutaryl-CoA reductase activity in cultured human fibroblasts by hypertriglyceridemic very low density lipoprotein subclasses.高甘油三酯血症极低密度脂蛋白亚类对培养的人成纤维细胞中3-羟基-3-甲基戊二酰辅酶A还原酶活性的异常抑制。

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Suppression of 3-hydroxy-3-methylglutaryl-CoA reductase by low density lipoproteins produced in vitro by lipoprotein lipase action on nonsuppressive very low density lipoproteins.

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Correlation between two effects of high density lipoproteins on vascular endothelial cells. The induction of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity and the support of cellular proliferation.高密度脂蛋白对血管内皮细胞两种作用之间的相关性。3-羟基-3-甲基戊二酰辅酶A还原酶活性的诱导及细胞增殖的支持。

J Biol Chem. 1982 Aug 25;257(16):9429-37.

[Cholesterol and triglyceride content of lipoproteins in hyperlipoproteinemia type II/a, II/b and IV].[II/a型、II/b型和IV型高脂蛋白血症中脂蛋白的胆固醇和甘油三酯含量]

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Effects of human low-density lipoproteins on the nucleotide patterns of cultured pig aortic endothelial cells.人低密度脂蛋白对培养的猪主动脉内皮细胞核苷酸模式的影响。

FEBS Lett. 1991 Sep 2;289(1):51-3. doi: 10.1016/0014-5793(91)80906-j.

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Role of the clinical biochemistry laboratory in the assessment of dyslipoproteinaemias.临床生物化学实验室在血脂异常评估中的作用。

Ann Clin Biochem. 1984 May;21 ( Pt 3):166-75. doi: 10.1177/000456328402100302.

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Clin Cardiol. 1999 Jun;22(6 Suppl):II7-14. doi: 10.1002/clc.4960221403.

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Am J Pathol. 1982 May;107(2):186-90.

Hypertriglyceridemic very low density lipoproteins induce triglyceride synthesis and accumulation in mouse peritoneal macrophages.高甘油三酯血症性极低密度脂蛋白诱导小鼠腹腔巨噬细胞中甘油三酯的合成与积累。

J Clin Invest. 1982 Jul;70(1):168-78. doi: 10.1172/jci110590.

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Fish and coronary artery disease.鱼类与冠状动脉疾病

Br Heart J. 1987 Mar;57(3):214-9. doi: 10.1136/hrt.57.3.214.

Oxidative modification of low density lipoprotein (LDL) by activated human monocytes and the cell lines U937 and HL60.活化的人单核细胞以及U937和HL60细胞系对低密度脂蛋白（LDL）的氧化修饰

In Vitro Cell Dev Biol. 1988 Oct;24(10):1001-8. doi: 10.1007/BF02620873.

Lipolytic surface remnants of triglyceride-rich lipoproteins are cytotoxic to macrophages but not in the presence of high density lipoprotein. A possible mechanism of atherogenesis?富含甘油三酯的脂蛋白的脂解表面残余物对巨噬细胞具有细胞毒性，但在高密度脂蛋白存在时则无此毒性。这是动脉粥样硬化形成的一种可能机制吗？

J Clin Invest. 1989 Apr;83(4):1363-74. doi: 10.1172/JCI114024.

Triglyceride-rich lipoproteins and atherosclerosis.富含甘油三酯的脂蛋白与动脉粥样硬化。

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