Henrichs K J, Berry C L
Virchows Arch A Pathol Anat Histol. 1980;385(3):351-9. doi: 10.1007/BF00432543.
Vessels of known position in the vascular tree of the kidneys of two cases with a long history of progressive systemic sclerosis--one normotensive, one hypertensive--were examined morphometrically. Medial thickness, intimal thickness and the relative content of collagen and elastin in the vascular media were measured. Smooth muscle nuclei were counted in the arterial cross section. These morphometric data were compared with those obtained from two autopsy cases--one with a history of essential hypertension, one without any hypertensive history. The findings suggest that progressive sclerosis induces intimal thickening in all branches of the renal artery down to a distented diameter of 200 microns. In the case where progressive sclerosis was complicated by arterial hypertension increased medial thicknesses were found, similar to the findings in the case with a history of essential hypertension.
对两例患有长期进行性系统性硬化症的患者（一例血压正常，一例高血压）肾脏血管树中已知位置的血管进行了形态计量学检查。测量了血管中层厚度、内膜厚度以及血管中层中胶原蛋白和弹性蛋白的相对含量。在动脉横截面上对平滑肌细胞核进行计数。将这些形态计量学数据与从两例尸检病例（一例有原发性高血压病史，一例无任何高血压病史）获得的数据进行比较。研究结果表明，进行性硬化会导致肾动脉所有分支直至直径扩张到200微米处的内膜增厚。在进行性硬化并发动脉高血压的病例中，发现中层厚度增加，这与有原发性高血压病史的病例中的发现相似。
