Sobieva Z I, Karpova M N
Biull Eksp Biol Med. 1980 Oct;90(10):405-7.
Effect of chemical desympathization on heart contractile function was studied in experiments on Wistar rats (22 experimental and 20 control animals) under conditions of exposure to functional loads. These loads were realized by heart stimulation at an increasing rate and by a short-term aorta occlusion (as maximum load). The data obtained indicate the depression of contractile function of the desympathized heart exposed to stimulation and maintenance of its contractility under occlusion. It is inferred that the adaptation resources of the desympathized heart are reduced under conditions that require mobilization of the mechanism [interval -- force] and are preserved during heart hyperfunction in isometric regime.
在功能性负荷条件下，对Wistar大鼠（22只实验动物和20只对照动物）进行实验，研究化学性去交感神经对心脏收缩功能的影响。这些负荷通过以递增速率刺激心脏和短期阻断主动脉（作为最大负荷）来实现。所获得的数据表明，去交感神经的心脏在受到刺激时收缩功能受到抑制，而在阻断时其收缩性得以维持。据推测，在需要调动[间期 - 力量]机制的条件下，去交感神经心脏的适应资源减少，而在等长状态下心脏功能亢进时则得以保留。
