α-银环蛇毒素对哺乳动物运动终板处由锋电位诱导的乙酰胆碱释放显然没有影响。

Apparent lack of effect on alpha-bungarotoxin on the spike-induced release of acetylcholine at the mammalian motor end-plate.

作者信息

Gelsema A J

出版信息

Neurosci Lett. 1980 Nov;20(2):189-93. doi: 10.1016/0304-3940(80)90144-5.

Abstract

During intoxication with alpha-bungarotoxin (alpha-BGT) the amplitude of end-plate potentials (EPPs) decreased more slowly than the amplitude of potentials evoked by iontophoretically applied acetylcholine. Mean quantal content of EPPs, however, remained constant. The results do not support the notion that alpha-BGT influences, the spike-evoked transmitter release.

摘要

在被α-银环蛇毒素（α-BGT）中毒期间，终板电位（EPPs）的幅度下降速度比离子电渗法施加乙酰胆碱所诱发电位的幅度下降速度更慢。然而，EPPs的平均量子含量保持不变。这些结果不支持α-BGT影响由锋电位诱发的递质释放这一观点。