耳廓软骨细胞的原位老化并非由于其复制潜能的耗尽。
In situ aging of auricular chondrocytes is not due to the exhaustion of their replicative potential.
作者信息
Moskalewski S, Gołaszewska A, Ksiazek T
出版信息
Experientia. 1980 Nov 15;36(11):1294-5. doi: 10.1007/BF01969596.
PMID:7449915
Abstract
Multiplication of chondrocytes during growth of rabbit auricular cartilage was estimated on the basis of total DNA determination and compared with the population doubling level reached by these chondrocytes in vitro. The results indicate that the in situ aging of auricular chondrocytes is caused by factors other than the intrinsic depletion of their growth potential.
摘要
基于总DNA测定评估了兔耳软骨生长过程中软骨细胞的增殖情况，并将其与这些软骨细胞在体外达到的群体倍增水平进行了比较。结果表明，耳软骨细胞的原位老化是由其生长潜能的内在耗竭以外的因素引起的。
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