Immunopathologic alterations of lymphatic tissues of mice infected with lymphocytic choriomeningitis virus. II. Pathogenetic mechanism.
作者信息
Lehmann-Grube F, Löhler J
出版信息
Lab Invest. 1981 Mar;44(3):205-13.
PMID:7464046
Abstract
摘要
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[Murine lymphocytic choriomeningitis: pathology of the lymphatic tissue].
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Immunopathological alterations of lymphatic tissues in mice infected with lymphocytic choriomeningitis virus.
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Murine hepatitis caused by lymphocytic choriomeningitis virus. I. The hepatic lesions.淋巴细胞性脉络丛脑膜炎病毒引起的小鼠肝炎。I. 肝脏病变
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[Lymphatic tissues of mice infected with lymphocytic choriomeningitis virus].[感染淋巴细胞性脉络丛脑膜炎病毒的小鼠的淋巴组织]
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The immunological system and the development of central nervous system lesions in acute viral infections.急性病毒感染中的免疫系统与中枢神经系统病变的发展
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引用本文的文献
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References.参考文献。
Perspect Med Virol. 1986;2:209-245. doi: 10.1016/S0168-7069(08)70043-0. Epub 2008 May 29.
2
Antiviral immune responses of lymphocytic choriomeningitis virus-infected mice lacking CD8+ T lymphocytes because of disruption of the beta 2-microglobulin gene.由于β2-微球蛋白基因缺失而缺乏CD8 + T淋巴细胞的淋巴细胞性脉络丛脑膜炎病毒感染小鼠的抗病毒免疫反应
J Virol. 1993 Jan;67(1):332-9. doi: 10.1128/JVI.67.1.332-339.1993.
3
In vivo replication of pathogenic and attenuated strains of Junin virus in different cell populations of lymphatic tissue.胡宁病毒致病株和减毒株在淋巴组织不同细胞群体中的体内复制
Infect Immun. 1983 Sep;41(3):1279-83. doi: 10.1128/iai.41.3.1279-1283.1983.
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Immunosuppression-induced susceptibility of inbred hamsters (Mesocricetus auratus) to lethal-disease by lymphocytic choriomeningitis virus infection.免疫抑制诱导近交系仓鼠（金黄地鼠）对淋巴细胞性脉络丛脑膜炎病毒感染导致的致死性疾病的易感性。
Arch Virol. 1987;97(1-2):61-76. doi: 10.1007/BF01310734.
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Lymphocytic choriomeningitis virus-induced disease of the central nervous system and the "antigen-sink" hypothesis.淋巴细胞性脉络丛脑膜炎病毒引起的中枢神经系统疾病与“抗原库”假说
Med Microbiol Immunol. 1986;175(2-3):205-8. doi: 10.1007/BF02122452.
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Virus-induced delayed-type hypersensitivity reaction is sequentially mediated by CD8+ and CD4+ T lymphocytes.病毒诱导的迟发型超敏反应依次由CD8 +和CD4 + T淋巴细胞介导。
Proc Natl Acad Sci U S A. 1989 May;86(9):3291-5. doi: 10.1073/pnas.86.9.3291.
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