Biochemical evidence of decreased muscarinic cholinergic neuronal communication following amygdala-kindled seizures.
作者信息
Dasheiff R M, Byrne M C, Patrone V, McNamara J O
出版信息
Brain Res. 1981 Feb 9;206(1):233-8. doi: 10.1016/0006-8993(81)90123-2.
PMID:7470892
Abstract
摘要
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Biochemical evidence of decreased muscarinic cholinergic neuronal communication following amygdala-kindled seizures.杏仁核点燃癫痫发作后毒蕈碱胆碱能神经元通讯减少的生化证据。
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Transient increase in muscarinic acetylcholine receptor and acetylcholinesterase in visual cortex on first exposure of dark-reared rats to light.首次将黑暗饲养的大鼠暴露于光线下时，视皮层中毒蕈碱型乙酰胆碱受体和乙酰胆碱酯酶的短暂增加。
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Muscarinic cholinergic receptors participate in the kindling model of epilepsy.毒蕈碱型胆碱能受体参与癫痫的点燃模型。
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