Blanch L, Baigorri F, Fernández R, Saura P, Vallés J, Joseph D, Artigas A
Servicio de Medicina Intensiva, Hospital de Sabadell, Barcelona.
Med Clin (Barc). 1995 Sep 9;105(7):261-3.
The increase in pulmonary vascular resistences in acute pulmonary thromboembolism (APT) is the consequence of anatomical obstruction and pulmonary artery constriction. The administration of inhaled nitric oxide (NO) may be therapeutically useful in acute pulmonary hypertension by APT given its limited vasodilator effect on pulmonary circulation. A patient with APT in whom this selective vasodilator effect was observed is presented. The authors suggest that the administration of inhaled NO may be a potentially beneficial coadjuvant therapy in acute pulmonary hypertension induced by APT.
急性肺血栓栓塞症（APT）时肺血管阻力增加是解剖学阻塞和肺动脉收缩的结果。吸入一氧化氮（NO）给药因其对肺循环的血管扩张作用有限，可能对APT所致的急性肺动脉高压具有治疗作用。本文报告了1例观察到这种选择性血管扩张作用的APT患者。作者认为，吸入NO给药可能是APT所致急性肺动脉高压的一种潜在有益的辅助治疗方法。
