Asbestos and mesothelioma: genetic lessons from a tragedy.
作者信息
Knudson A
机构信息
Institute for Cancer Research, Fox Chase Cancer Center, Philadelphia, PA 19111, USA.
出版信息
Proc Natl Acad Sci U S A. 1995 Nov 21;92(24):10819-20. doi: 10.1073/pnas.92.24.10819.
Abstract
摘要
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本文引用的文献
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RELATION BETWEEN EXPOSURE TO ASBESTOS AND MESOTHELIOMA.接触石棉与间皮瘤之间的关系。
N Engl J Med. 1965 Mar 18;272:560-5. doi: 10.1056/NEJM196503182721104.
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A novel moesin-, ezrin-, radixin-like gene is a candidate for the neurofibromatosis 2 tumor suppressor.一种新的类肌动蛋白结合蛋白、埃兹蛋白、根蛋白样基因是2型神经纤维瘤病肿瘤抑制基因的候选基因。
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The Wilms tumour gene WT1 is expressed in murine mesoderm-derived tissues and mutated in a human mesothelioma.威尔姆斯瘤基因WT1在小鼠中胚层来源的组织中表达，并在一种人类间皮瘤中发生突变。
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Physical and functional interaction between WT1 and p53 proteins.WT1蛋白与p53蛋白之间的物理和功能相互作用。
Proc Natl Acad Sci U S A. 1993 Jun 1;90(11):5100-4. doi: 10.1073/pnas.90.11.5100.
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Alteration in a new gene encoding a putative membrane-organizing protein causes neuro-fibromatosis type 2.一种编码假定膜组织蛋白的新基因的改变会导致2型神经纤维瘤病。
Nature. 1993 Jun 10;363(6429):515-21. doi: 10.1038/363515a0.
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Recurrent deletions of specific chromosomal sites in 1p, 3p, 6q, and 9p in human malignant mesothelioma.人类恶性间皮瘤中1p、3p、6q和9p特定染色体位点的反复缺失
Cancer Res. 1993 Sep 15;53(18):4349-55.
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Rates of p16 (MTS1) mutations in primary tumors with 9p loss.伴有9号染色体短臂缺失的原发性肿瘤中p16（MTS1）基因突变率。
Science. 1994 Jul 15;265(5170):415-7. doi: 10.1126/science.8023167.
8
p16 alterations and deletion mapping of 9p21-p22 in malignant mesothelioma.恶性间皮瘤中9p21 - p22的p16改变与缺失定位
Cancer Res. 1994 Nov 1;54(21):5547-51.
9
Neurofibromatosis type 2 (NF2) gene is somatically mutated in mesothelioma but not in lung cancer.2型神经纤维瘤病（NF2）基因在间皮瘤中发生体细胞突变，但在肺癌中未发生。
Cancer Res. 1995 Mar 15;55(6):1227-31.
10
G1 phase arrest induced by Wilms tumor protein WT1 is abrogated by cyclin/CDK complexes.由肾母细胞瘤蛋白WT1诱导的G1期阻滞被细胞周期蛋白/细胞周期蛋白依赖性激酶复合物消除。
Proc Natl Acad Sci U S A. 1995 May 9;92(10):4517-21. doi: 10.1073/pnas.92.10.4517.
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