Kolb M, Petersen D, Schiefer U, Kolb R, Skalej M
Department of Pathophysiology of Vision and Neuro-Ophthalmology, University Eye Hospital, Tübingen, Germany.
Ger J Ophthalmol. 1995 Jul;4(4):228-33.
The authors examined 59 patients (32 women and 27 men) suffering from homonymous hemianopia by white-noise-field campimetry (Tübingen Electronic Campimeter) and by conventional perimetry (Tübingen Automatic Perimeter or Tübingen Manual Perimeter) and by computerized tomography (CT) or magnetic resonance imaging (MR). In all, 56 patients showed neuroradiologically detectable cerebral lesions, which were superimposed in a reconstructed "reference brain". Of these 56 patients, 18 were not capable of perceiving any white-noise-field scotoma, although their conventional perimetric findings showed defects. Lesions of these patients were concentrated along the optic radiation and spared the primary visual cortex. The lesions of the remaining 38 patients with scotoma detectable in both methods were accumulated in area V1, the primary visual cortex. The lesions of 6 patients, who had had persistent scotoma perception for more than 2 years, were situated at the occipital pole. The lesions of 8 patients with vanishing scotoma were concentrated in more rostral areas.
作者通过白噪声视野计（图宾根电子视野计）、传统视野计（图宾根自动视野计或图宾根手动视野计）以及计算机断层扫描（CT）或磁共振成像（MR）对59例患有同向偏盲的患者（32名女性和27名男性）进行了检查。总共有56例患者显示出神经放射学可检测到的脑部病变，这些病变被叠加在一个重建的“参考脑”中。在这56例患者中，18例尽管传统视野检查结果显示有缺损，但却无法察觉到任何白噪声视野暗点。这些患者的病变集中在视辐射沿线，而初级视皮层未受影响。其余38例在两种检查方法中均能检测到暗点的患者，其病变集中在V1区，即初级视皮层。6例有持续暗点感知超过2年的患者，其病变位于枕极。8例暗点逐渐消失的患者，其病变集中在更靠前的区域。
