Kuroiwa T, Tanabe H, Takatsuka H, Arai M, Ozaki T, Nagasawa S, Ohta T
Department of Neurosurgery, Osaka Mishima Critical Care Medical Center.
Neurol Med Chir (Tokyo). 1994 Jan;34(1):20-2. doi: 10.2176/nmc.34.20.
A 19-year-old male was admitted following a blow to the face. Computed tomographic (CT) scans 1 hour after injury revealed low-density areas in the bilateral thalami and midbrain, which were enhanced postcontrast except for the core 3 hours later. CT scans 2 days after injury revealed that the size of the low-density areas had increased. CT scans and magnetic resonance images 3 weeks after injury disclosed only small infarcted lesions in the bilateral thalami, the right side of the midbrain, and the left internal capsule. These findings suggest that the injury initially caused thrombus on the basilar arterial wall, leading to occlusion of the perforators, but almost all affected perforators were recanalized. Bilateral thalamic infarction resulting from head injury is unusual, as is the transient nature of the infarction in this case.
一名19岁男性在面部遭受打击后入院。受伤后1小时的计算机断层扫描(CT)显示双侧丘脑和中脑有低密度区,3小时后除核心区域外,造影后强化。受伤后2天的CT扫描显示低密度区面积增大。受伤后3周的CT扫描和磁共振成像仅显示双侧丘脑、中脑右侧和左侧内囊有小梗死灶。这些发现表明,损伤最初导致基底动脉壁血栓形成,导致穿支闭塞,但几乎所有受影响的穿支都再通了。头部损伤导致的双侧丘脑梗死并不常见,本例梗死的短暂性也不常见。