FK506和雷帕霉素对白细胞介素1β及免疫球蛋白体外产生的直接作用。 - Suppr | 超能文献

The direct effect of FK506 and rapamycin on interleukin 1(beta) and immunoglobulin production in vitro.

作者信息

Yoshimura N, Ohmoto Y, Yasui H, Ohsaka Y, Bong J M, Kobayashi Y, Oka T

机构信息

Department of Surgery, Kyoto Prefectural University of Medicine, Japan.

出版信息

Transplantation. 1994 Jun 27;57(12):1815-8.

Abstract

摘要

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The direct effect of FK506 and rapamycin on interleukin 1(beta) and immunoglobulin production in vitro.FK506和雷帕霉素对白细胞介素1β及免疫球蛋白体外产生的直接作用。

Transplantation. 1994 Jun 27;57(12):1815-8.

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Effect of cyclosporine on interleukin 1(beta) and immunoglobulin production in vitro.环孢素对白细胞介素1（β）及免疫球蛋白体外产生的影响。

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Comparative immunoregulatory effects of rapamycin, FK 506 and cyclosporine on mitogen-induced cytokine production and lymphoproliferation.雷帕霉素、FK 506和环孢素对丝裂原诱导的细胞因子产生和淋巴细胞增殖的比较免疫调节作用。

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Effect of rapamycin on the in vitro release of soluble interleukin-2 receptor by phytohemagglutinin, phorbol myristate acetate, and ionomycin-activated peripheral blood mononuclear cells.雷帕霉素对植物血凝素、佛波酯和离子霉素激活的外周血单个核细胞体外释放可溶性白细胞介素-2受体的影响。

Transplant Proc. 1995 Feb;27(1):432-4.

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Modes of action of FK506, cyclosporin A, and rapamycin.FK506、环孢素A和雷帕霉素的作用模式。

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FK 506, rapamycin, and cyclosporine: effects on IL-4 and IL-10 mRNA levels in a T-helper 2 cell line.

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The hydroxylamine of sulfamethoxazole synergizes with FK506 and cyclosporin A, inhibiting T-cell proliferation.磺胺甲恶唑的羟胺与FK506和环孢素A协同作用，抑制T细胞增殖。

J Pharmacol Exp Ther. 1997 Apr;281(1):540-8.

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FK506 as an agonist to induce inhibition of interleukin 2 production.

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Transforming growth factor beta 1 inhibits interleukin-1-induced but enhances ionomycin-induced interferon-gamma production in a T cell lymphoma: comparison with the effects of rapamycin.转化生长因子β1抑制白细胞介素-1诱导的，但增强离子霉素诱导的T细胞淋巴瘤中干扰素-γ的产生：与雷帕霉素的作用比较。

J Cell Physiol. 1994 Jul;160(1):141-53. doi: 10.1002/jcp.1041600117.

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FK506 potently inhibits T cell activation induced TNF-alpha and IL-1beta production in vitro by human peripheral blood mononuclear cells.FK506能有效抑制人外周血单个核细胞在体外诱导的T细胞活化及肿瘤坏死因子-α（TNF-α）和白细胞介素-1β（IL-1β）的产生。

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