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[血管壁收缩的“非肌动球蛋白”成分]

[The "non-actomyosin" component of vascular wall contraction].

作者信息

Shapovalov A N, Shuba M F

出版信息

Fiziol Zh Im I M Sechenova. 1994 Feb;80(2):59-67.

PMID:7522787
Abstract

The matrix of connective tissue was found to take part in generation of the mechanical strength in isolated strips of the v. cava posterior wall under the effect of increased temperature. The finding corroborates the concept of the actomyosin interaction. The vessel tissue response to temperature seems to be formed by three mechanisms, two of them being of a non-actomyosin nature.

摘要

研究发现,在温度升高的影响下,结缔组织基质参与了下腔静脉后壁分离条带机械强度的产生。这一发现证实了肌动球蛋白相互作用的概念。血管组织对温度的反应似乎由三种机制形成,其中两种机制是非肌动球蛋白性质的。

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