Schäfer W, Sattler R
Chir Forum Exp Klin Forsch. 1978(1978):107-11.
Arterial and venous [K+] and [H+] concentrations were determined during normothermic ischemic arrest in cat hearts intermittently perfused without resuscitation. The slight loss in [K+] was taken as evidence of a stable membrane potential. The decrease in [H+] activity appearing in the perfusate correlated well with the previously described increase in coronary resistance. This was interpreted as a sign of restricted coronary perfusion and not as evidence for reduced anaerobic metabolic activity. Thus the functional impairment of the coronary system was confirmed to be a limiting factor when resuscitating ischemically arrested hearts.
在未进行复苏的情况下，对间歇性灌注的猫心脏进行常温缺血性停搏期间，测定动脉和静脉的[K⁺]和[H⁺]浓度。[K⁺]的轻微降低被视为膜电位稳定的证据。灌注液中出现的[H⁺]活性降低与先前描述的冠状动脉阻力增加密切相关。这被解释为冠状动脉灌注受限的迹象，而不是无氧代谢活性降低的证据。因此，冠状动脉系统的功能损害被确认为复苏缺血性停搏心脏时的一个限制因素。
