Meyer-Franke A, Barres B
Stanford University School of Medicine, Department of Neurobiology, California 94305-5401.
Curr Biol. 1994 Sep 1;4(9):847-50. doi: 10.1016/s0960-9822(00)00190-1.
Mice lacking myelin-associated glycoprotein surprisingly myelinate almost normally but their oligodendrocytes have lost their periaxonal cytoplasmic 'collars' and accidentally myelinate already-myelinated axons.
缺乏髓鞘相关糖蛋白的小鼠出人意料地几乎能正常形成髓鞘，但它们的少突胶质细胞失去了轴突周围的细胞质“套环”，并意外地对已经形成髓鞘的轴突再次进行髓鞘形成。
