Buchta R M, Bickerton R
Division of Pediatrics/Adolescent Medicine, Scripps Clinic and Research Foundation, La Jolla, California 92037.
J Adolesc Health. 1994 Nov;15(7):592-4. doi: 10.1016/1054-139x(94)90144-r.
The level of severe compensated iron deficiency anemia incompatible with life is not defined in the pediatric or adolescent literature. A hemoglobin of 1.5 gm/dl in an older adolescent with few physical symptoms is distinctly unusual. A case of profound iron deficiency anemia in a 20-year-old developmentally delayed male is the subject of this brief report. There were only subtle physical findings in spite of this severe anemia. The anemia was the result of corrosive esophagitis associated with a hiatal hernia and reflux. Physicians dealing with developmentally delayed adolescents should be aware of the fact that a severe anemia may develop, and such individuals should be periodically screened for anemia, melena, hematochezia, and hematemesis.
小儿或青少年医学文献中未明确界定与生命不相容的严重代偿性缺铁性贫血水平。一名身体症状较少的大龄青少年血红蛋白水平为1.5克/分升，这显然不寻常。本简要报告的主题是一名20岁发育迟缓男性的严重缺铁性贫血病例。尽管存在这种严重贫血，但体格检查结果仅有细微异常。贫血是由与食管裂孔疝和反流相关的腐蚀性食管炎所致。治疗发育迟缓青少年的医生应意识到可能会发生严重贫血，并且应对此类个体定期进行贫血、黑便、便血和呕血筛查。
